t(5;12)(q31;p13)/ETV6::ACSL6 and t(6;9)(p23;q34)/DEK::NUP214 concurrence in acute myeloid leukemia: an unusual association of two rare abnormalities

Baldazzi, Carmen; Luatti, Simona; Marzocchi, Giulia; Grassi, Alessandra; Cavo, Michèle; Testoni, Nicoletta

doi:10.1016/j.cancergen.2021.12.006

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“…Previous literature supports the role of several of these genes as oncogenic drivers in AML. For instance, the ETV6ACSL6 rearrangement has been implicated in the pathogenesis of leukemia [ 30 ]. The activation of the MAP3K1 kinase has been implicated as a contributing factor in the pathogenesis of various malignant neoplasms, rendering it a prospective therapeutic target, particularly in the context of acute myeloid leukemia (AML) [ 31 ].…”

Section: Discussionmentioning

confidence: 99%