Systemic and Blood Findings in Parodontosis1

Goldstein, Harold M.

doi:10.1177/00220345380170010201

J Dent Res

1938

DOI: 10.1177/00220345380170010201

|View full text |Cite

Systemic and Blood Findings in Parodontosis1

Harold M. Goldstein

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...

Citation Types

Supporting

Mentioning

Contrasting

Year Published

1952

1982

Publication Types

Select...

Article5

Relationship

Self Cite0

Independent5

Authors

Journals

Cited by 5 publications

(1 citation statement)

References 0 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…These however are oral manifestations of systemic diseases and should not be confused with the condition that was described as idiopathic periodontosis. These diseases include diabetes (Goldstein 1938), sarcoidosis (van Swol 1973, Downs syndrome (Kisling & Krebs 1963, Saxen et al 1977. Papillon-Lefevre syndrome (Bouyssou & Fourel 1973) cyclic neutropenia (Cohen & Morris 1961), agranuiocytosis (Bauer 1946) and Chediak-Higashi disease (Tempel et al 1972).…”

mentioning

confidence: 99%

Juvenile periodontitis‐Healing following therapy to control inflammatory and traumatic etiologic components of the disease

Evian

Amsterdam

Rosenberg

1982

J Clinic Periodontology

View full text Add to dashboard Cite

A case of localized juvenile periodontitis with bone loss affecting molars and premolars with advanced destruction around the lower first molars is described. These lesions were associated with areas of excess occlusal trauma. The ramifications of combined therapy aimed at the elimination and control of both the inflammatory and occlusal components of the disease are discussed relative to the successful treatment of this case. A description of the periodontal status of a sibling of the patient has been included. Extensive defects are associated with the anterior teeth and in this case the patient presented with excessive trauma in the anterior region. Considering the localities of the disease and the similarities between the inflammatory components in both cases, a link between occlusal trauma and juvenile periodontitis is discussed.

show abstract

mentioning

confidence: 99%

Juvenile periodontitis‐Healing following therapy to control inflammatory and traumatic etiologic components of the disease

Evian

Amsterdam

Rosenberg

1982

J Clinic Periodontology

View full text Add to dashboard Cite

show abstract

Juvenile periodontitis

Saxén

1980

J Clinic Periodontology

View full text Add to dashboard Cite

Our knowledge of juvenile periodontitis is still fragmentary. In 50 years we have advanced from the concept of diffuse atrophy of the alveolar bone (Gottlieb 1923) through the theory of non-inflammatory, degenerative disease of the periodontium (Orban & Weinmann 1942) to the present conception of juvenile periodontitis (Manson & Lehner 1974, Waerhaug 1977a) as a periodontal disease appearing in young individuals with inflammation always present. Only the clinical picture of the disease is quite clear as Baer described it (1971): rapid destruction of the alveolar bone, not commensurate with the local irritants, around more than one permanent tooth in otherwise healthy adolescents. The etiology and etiopathogenesis of juvenile periodontitis have remained unknown. The bacteriological findings of Scransky et al. (1970) and Newman et al. (1974), suggesting some Gram-negative rods as an etiological factor, are still controversial. Neither is the theory of Lehner and his coworkers (1974), that juvenile periodontitis is a selective, cell-mediated immunodeficiency condition, fully accepted. Heredity is an etiologic factor for which there is more evidence. Several authors have found a familial pattern of the disease and it might be either an autosomal, recessive trait (Fourel 1972, Jorgenson et al. 1975) or an X-linked dominant disease (Melnick et al. 1976). These two statements are, however, also controversial. The prevalence rates vary from 0.1% to 17.6%. Although there could be racial variations, the estimated prevalence rates also vary within racial groups, suggesting that there must be great variations in methods and diagnostics. Juvenile periodontitis seems to exist in all racial groups throughout the world and although comparable prevalence figures are difficult to obtain, it seems that the disease is less common in Caucasoid populations and more frequent in India and Africa. The claim of female preponderence requires further investigation.

show abstract

Report of Evaluating Committee III Systemic Factors in Periodontal Disease

1952

The Journal of the American Dental Association

View full text Add to dashboard Cite

scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.

Contact Info

hi@scite.ai

10624 S. Eastern Ave., Ste. A-614

Henderson, NV 89052, USA

Blog Terms and Conditions API Terms Privacy Policy Contact Cookie Preferences Do Not Sell or Share My Personal Information

Made with 💙 for researchers

Part of the Research Solutions Family.

Systemic and Blood Findings in Parodontosis1

Cited by 5 publications

References 0 publications

Juvenile periodontitis‐Healing following therapy to control inflammatory and traumatic etiologic components of the disease

Juvenile periodontitis‐Healing following therapy to control inflammatory and traumatic etiologic components of the disease

Juvenile periodontitis

Report of Evaluating Committee III Systemic Factors in Periodontal Disease

Contact Info

Product

Resources

About