1988
DOI: 10.2214/ajr.150.2.391
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Syringomyelia as a consequence of compressive extramedullary lesions: postoperative clinical and radiological manifestations

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Cited by 32 publications
(10 citation statements)
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“…3 The transmedullary movement of cerebrospinal fluid may be caused by active transport across cell membranes or flow through dilated perivascular spaces; this would increase diffusion leading to the formation of intramedullary microcysts. 24 A reduced ADC lesion surrounded by an elevated ADC lesion may be caused by vascular compromise with cellular swelling as the initial change in the spinal cord. [25][26][27] Unexpectedly, our results demonstrated that the distribution of abnormalities in ADC maps and T2-weighted images differ greatly among patients.…”
Section: Discussionmentioning
confidence: 99%
“…3 The transmedullary movement of cerebrospinal fluid may be caused by active transport across cell membranes or flow through dilated perivascular spaces; this would increase diffusion leading to the formation of intramedullary microcysts. 24 A reduced ADC lesion surrounded by an elevated ADC lesion may be caused by vascular compromise with cellular swelling as the initial change in the spinal cord. [25][26][27] Unexpectedly, our results demonstrated that the distribution of abnormalities in ADC maps and T2-weighted images differ greatly among patients.…”
Section: Discussionmentioning
confidence: 99%
“…MR methods [4,[9][10][11] for the qualitative study of fluid movement are non-flow compensated T2W image and phase-shift imaging. Quantitative MR methods are phase-contrast cine MR and dual flip angle technique [5,8,9]. The methods of non-motion-compensated T2W sequence have a threshold for the detection of fluid movement.…”
Section: The Study Of Syringeal Fluid Motionmentioning
confidence: 99%
“…43,44 The fixation of the spinal cord by adhesions and arachnoiditis at the site of the lesion also appears to play a role. 4,19 The first symptom in most cases is pain, which is increased by straining, coughing or sneezing/ 27, 41 followed by sensory deficit, motor loss and increase of spasticity.29 Autonomous symptoms such as abnormal sweating or hypertension are less frequent,29 but can also be the first symptom. 37 The earliest clinical sign is loss of reflexes.…”
mentioning
confidence: 99%