2022
DOI: 10.1016/j.bioorg.2022.106142
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Synthesis and in vivo evaluation of new steviol derivatives that protect against cardiomyopathy by inhibiting ferroptosis

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Cited by 5 publications
(5 citation statements)
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“…Fer-1 can ameliorate TMEM43 knockdown-induced deteriorating effects in LPS-induced cardiac injury. Taken together, TMEM43 protects against SCM by inhibiting ferroptosis (Chen Z. et al, 2022). The islet cell autoantigen 69, which can regulate inflammation and immune response, induces ferroptosis to cause septic cardiac dysfunction through the stimulator of interferon gene trafficking (Kong et al, 2022).…”
Section: Ferroptosis In Septic Cardiomyopathymentioning
confidence: 96%
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“…Fer-1 can ameliorate TMEM43 knockdown-induced deteriorating effects in LPS-induced cardiac injury. Taken together, TMEM43 protects against SCM by inhibiting ferroptosis (Chen Z. et al, 2022). The islet cell autoantigen 69, which can regulate inflammation and immune response, induces ferroptosis to cause septic cardiac dysfunction through the stimulator of interferon gene trafficking (Kong et al, 2022).…”
Section: Ferroptosis In Septic Cardiomyopathymentioning
confidence: 96%
“…25 attenuates DICM through inhibiting ferroptosis in vivo and in vitro by limiting iron accumulation, restoring GPX4, and preventing LPO (Chen L. et al, 2022). AMPK inhibitor compound C reversed protective function of 25 against DICM, suggesting that 25 markedly downregulated ferroptosis by activating AMPKdependent signaling pathways (Chen Z. et al, 2022). Dexazoxane(26) , melatonin(27) (Sun et al, 2022), and EGCG(28) (He et al, 2021) also works as a GPX4 activators to attenuates DICM through inhibiting ferroptosis.…”
Section: Inhibiting Ferroptosis To Treat Dicmmentioning
confidence: 99%
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