Syndrome cave supérieur : une cause secondaire d’élévation de la pression intracrânienne après traumatisme crânien grave

Piteaud, I.; Abdennour, Lamine; Icke, Christian; Stany, Ida; Lescot, Thomas; Puybasset, Louis

doi:10.1016/j.annfar.2008.08.002

Cited by 2 publications

(1 citation statement)

References 22 publications

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“…The internal cerebral vein is generally used to evaluate the status of cerebral venous drainage. High-grade fluctuation in the internal cerebral vein was reported to be associated with IVH [ 46 ], and an increase in the preload, the diastolic pressure of the right ventricle, or intrathoracic pressure was determined to impede central venous return and increase in cerebral venous pressure [ 88 ]. Lower superior vena cava flow is caused by considerable shunting of hsPDA [ 10 ] and impaired myocardial function [ 56 ] and is associated with an increased risk of IVH [ 57 ].…”

Section: Altered Cerebral Haemodynamicsmentioning

confidence: 99%

Pathogenesis and Prevention of Intraventricular Hemorrhage in Preterm Infants

Tsao¹

2023

J Korean Neurosurg Soc

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Intraventricular haemorrhage (IVH) is a serious concern for preterm infants and can predispose such infants to brain injury and poor neurodevelopmental outcomes. IVH is particularly common in preterm infants. Although advances in obstetric management and neonatal care have led to a lower mortality rate for preterm infants with IVH, the IVH-related morbidity rate in this population remains high. Therefore, the present review investigated the pathophysiology of IVH and the evidence related to interventions for prevention. The analysis of the pathophysiology of IVH was conducted with a focus on the factors associated with cerebral hemodynamics, vulnerabilities in the structure of cerebral vessels, and host or genetic predisposing factors. The findings presented in the literature indicate that fluctuations in cerebral blood flow, the presence of hemodynamic significant patent ductus arteriosus, arterial carbon dioxide tension, and impaired cerebral venous drainage; a vulnerable or fragile capillary network; and a genetic variant associated with a mechanism underlying IVH development may lead to preterm infants developing IVH. Therefore, strategies focused on antenatal management, such as routine corticosteroid administration and magnesium sulfate use; perinatal management, such as maternal transfer to a specialized center; and postnatal management, including pharmacological agent administration and circulatory management involving prevention of extreme blood pressure, hemodynamic significant patent ductus arteriosus management, and optimization of cardiac function, can lower the likelihood of IVH development in preterm 4 infants. Incorporating neuroprotective care bundles into routine care for such infants may also reduce the likelihood of IVH development. The findings regarding the pathogenesis of IVH further indicate that cerebrovascular status and systemic hemodynamic changes must be analyzed and monitored in preterm infants and that individualized management strategies must be developed with consideration of the risk factors for and physiological status of each preterm infant.

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Section: Altered Cerebral Haemodynamicsmentioning

confidence: 99%

Pathogenesis and Prevention of Intraventricular Hemorrhage in Preterm Infants

Tsao¹

2023

J Korean Neurosurg Soc

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Brain swelling in acute superior vena cava syndrome due to aortic dissection: unusual and lethal manifestation aggravated by induction of general anesthesia

Koizumi

Ueda

Hashizume

et al. 2012

Gen Thorac Cardiovasc Surg

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A 32-year-old woman with Marfan syndrome experienced acute superior vena cava syndrome due to aortic dissection. The patient had previously undergone a Bentall operation. The aneurysm from the ascending to the transverse aorta compressed the superior vena cava, the right pulmonary artery, and the trachea. The rare and life-threatening neurological complication in this patient may have been related to brain edema, which was revealed by preoperative computed tomography. The induction of general anesthesia aggravated the symptoms of the superior vena cava syndrome and led to a fatal condition. Additional cannulation in the right subclavian vein was mandatory to alleviate the symptoms because the venous drainage from the upper half of the body created only by the femorofemoral bypass was not adequate. Total arch replacement was performed. The postoperative course was uneventful.

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Syndrome cave supérieur : une cause secondaire d’élévation de la pression intracrânienne après traumatisme crânien grave

Cited by 2 publications

References 22 publications

Pathogenesis and Prevention of Intraventricular Hemorrhage in Preterm Infants

Pathogenesis and Prevention of Intraventricular Hemorrhage in Preterm Infants

Brain swelling in acute superior vena cava syndrome due to aortic dissection: unusual and lethal manifestation aggravated by induction of general anesthesia

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