Symptomatic Vertebral Artery Stenosis Secondary to Cervical Spondylolisthesis

Piñol, Ignasi; Valencia, Manuel Ramírez; Saló, Guillem; Ros, Antoni Molina; Blanch, Andreu Lladó

doi:10.1097/brs.0b013e3182a43441

Cited by 17 publications

(8 citation statements)

References 12 publications

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“…Our case did not show above features but occlusion of vertebral artery was limited in transverse foramen of atlas. This bow hunter's syndrome was difficult to diagnose without suspicion of cervical spine lesion of vertebral artery 2 , 4 , 9) .…”

Section: Discussionmentioning

confidence: 94%

“…Numerous causes of VBI are reported of which the most common cause is arthrosclerosis of the arteries distributed in the brain stem 1) . Osteophytes, cervical musculature, fascial bands, anomalies of cervical spine and vertebral artery are other extracranial mechanical causes of VBI 1 , 9) . Most of reported cases in bow hunter's syndrome showed occlusion of a dominant vertebral artery due to mechanical stretching or compression and dislodgment between C1-2 with head rotation 8) .…”

Section: Discussionmentioning

confidence: 99%

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Cervicogenic Vertigo Treated by C1 Transverse Foramen Decompression : A Case Report

et al. 2014

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Cervicogenic vertigo was known as Bow hunter's syndrome. Occlusion of vertebral artery causes vertebrobasilar insufficiency and we reported cervicogenic vertigo case which was treated by simple decompression of transverse foramen of C1. The patient was 48 years old female who had left side dominant vertebral artery and vertigo was provoked when she rotated her head to right side. Angiography showed complete obliteration of blood flow of left vertebral artery when her head was rotated to right side. The operation was decompression of left vertebral artery at C1 level. Posterior wall of transverse foramen was resected and vertebral artery was exposed and decompressed. After surgery, vertigo of the patient was disappeared, and angiography showed patent left vertebral artery when her head was rotated to right side. Vertigo caused by compression of cervical vertebral artery could be treated by decompression without fusion or instrumentation, especially in C1 transverse foramen.

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Section: Discussionmentioning

confidence: 94%

Section: Discussionmentioning

confidence: 99%

Cervicogenic Vertigo Treated by C1 Transverse Foramen Decompression : A Case Report

et al. 2014

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“… 8 , 9) Other less frequent causes include osteophytes of the uncovertebral joint, disc herniation, and spondylolisthesis at the C3–6 level where the VA passes through the transverse foramen (V2 segment). 1 – 3 , 10 , 11) Neurological symptoms of V2 RVAS are provoked by ipsilateral head rotation or neck extension in most cases, contrary to classic RVAS at the C1/2 level. To the best of our knowledge, a spinal tumor has never been implicated as the cause of RVAS in the V2 segment.…”

Section: Discussionmentioning

confidence: 99%

Surgical Treatment of Rotational Vertebral Artery Syndrome Induced by Spinal Tumor: A Case Report and Literature Review

Haimoto

Nishimura

Hara

et al. 2017

NMC Case Report Journal

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Vertebrobasilar insufficiency (VBI) provoked by physiological head rotation is known as rotational vertebral artery syndrome (RVAS) or Bow Hunter syndrome. RVAS most often occurs at C1–2 level with head rotation and presents with symptoms of VBI. Several previously published studies have reported RVAS at subaxial sites (V2 segment), however, tumor-induced RVAS has never been reported. The authors report the first case of RVAS at V2 segment due to compression from a spinal tumor. A 71-year-old man presented with symptoms of dizziness provoked by head rotation or neck extension. computed tomography (CT) angiography and dynamic cerebral angiography revealed circumferential stenosis with neutral neck position and complete occlusion of the left dominant vertebral artery (VA) at C5 level with his neck extended or rotated to the left. Complete neurological recovery was achieved after removal of a spinal osteochondroma and surgical decompression of the left VA via an anterior approach. Spinal tumors should be included in the differential diagnosis in cases of RVAS. Spinal degenerations or sarcomatous transformation of the tumor could lead to clinical manifestations of RVAS in cases with spinal osteochondroma. Complete removal of the tumor with or without spinal fusion would be the treatment of choice, in addition to medical treatment in the cases of acute stroke.

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“…Симптоматика в каждом конкретном случае определяется анатомией задней системы циркуляции и патологией шейных структур. Так, сочетание гипоплазии ПА, несостоятельности гомолатеральной задней соединительной артерии и компрессии гетеролатеральной гемодинамически доминирующей ПА повышает вероятность развития стволового инсульта [35].…”

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Cervical vertigo: a neurologist's point of view

Stulin

Tardov

Kunelskaya

et al. 2018

Z. nevrol. psikhiatr. im. S.S. Korsakova

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Cervical vertigo (CV) hasn't yet been recognized by many neurologists, as it has no specific clinical symptoms and reliable diagnostic criteria. However, over the past 150 years, there has been accumulated numerous experimental and clinical evidence to support the legitimacy of CV allocation as a separate nosology. This review presents data regarding the vertigo associated with neck pathology: possible evidence of CV existence and the description of its type as well. The principles of CV diagnosis and treatment are considered, an attempt to create a unified concept is proposed.

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Symptomatic Vertebral Artery Stenosis Secondary to Cervical Spondylolisthesis

Abstract: Isthmic spondylolisthesis must be considered as a cause within VBI. Surgery presents good results.

Cited by 17 publications

References 12 publications

Cervicogenic Vertigo Treated by C1 Transverse Foramen Decompression : A Case Report

Cervicogenic Vertigo Treated by C1 Transverse Foramen Decompression : A Case Report

Surgical Treatment of Rotational Vertebral Artery Syndrome Induced by Spinal Tumor: A Case Report and Literature Review

Cervical vertigo: a neurologist's point of view

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