Surgical treatment of ventricular septal defect complicating acute myocardial infarction. Experience of a north Italian referral hospital

G, Cerin; Donato, Marisa Di; Dimulescu, Doina; Montericcio, Vincenzo; Menicanti, Lorenzo; Frigiola, Alessandro; Ambroggi, L. De

doi:10.1016/s0967-2109(02)00190-4

Cited by 52 publications

(41 citation statements)

References 21 publications

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“…The reason for the same is not clear as almost all our patients had preserved RV function. LV function in our group of patients was better than reported in other series [11].…”

contrasting

confidence: 62%

Ventricular septal rupture following myocardial infarction: Clinical, haemodynamic, angiographic profile and long-term outcome

Harikrishnan

Tharakan

Titus

et al. 2007

International Journal of Cardiology

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“…The reason for the same is not clear as almost all our patients had preserved RV function. LV function in our group of patients was better than reported in other series [11].…”

contrasting

confidence: 62%

Ventricular septal rupture following myocardial infarction: Clinical, haemodynamic, angiographic profile and long-term outcome

Harikrishnan

Tharakan

Titus

et al. 2007

International Journal of Cardiology

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“…4 This introduces a significant selection bias into surgical series, artificially inflating survival rates. [5][6][7][8] The advent of the Amplatzer family of ventricular septal defect (VSD) closure devices offers a potentially attractive alternative to surgical repair with multiple device implants possible if required ( Figure 1A). A few series of selected cases exist, with good results reported, [9][10][11] but there are no large series with early as well as delayed intervention.…”

mentioning

confidence: 99%

Percutaneous Closure of Postinfarction Ventricular Septal Defect

et al. 2014

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Background-Postinfarction ventricular septal defect carries a grim prognosis. Surgical repair offers reasonable outcomes in patients who survive a healing phase. Percutaneous device implantation represents a potentially attractive early alternative. Methods and Results-Postinfarction ventricular septal defect closure was attempted in 53 patients from 11 centers (1997)(1998)(1999)(2000)(2001)(2002)(2003)(2004)(2005)(2006)(2007)(2008)(2009)(2010)(2011)(2012); aged 72±11 years; 42% female). Nineteen percent had previous surgical closure. Myocardial infarction was anterior (66%) or inferior (34%). Time from myocardial infarction to closure procedure was 13 (first and third quartiles, 5-54) days. Devices were successfully implanted in 89% of patients. Major immediate complications included procedural death (3.8%) and emergency cardiac surgery (7.5%). Immediate shunt reduction was graded as complete (23%), partial (62%), or none (15%). Median length of stay after the procedure was 5.0 (2.0-9.0) days. Fifty-eight percent survived to discharge and were followed up for 395 (63-1522) days, during which time 4 additional patients died (7.5%).

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“…In the report of 189 patients from Sweden, several factors were able to predictable favorable vs unfavorable short (<30 days) and long-term (>30) outcomes (Tables 2 and 3) (Jeppsson, 2005). In a similar national registry experience, Cerin and colleagues reported the outcomes in 58 patients treated with PI-VSD from 1992 to 2000 in Italy (Cerin, 2003). The mean age was 73 years/old.…”

Section: Outcomes: Predictors Of Survivalmentioning

confidence: 99%

Post Myocardial Infarction Ventricular Septal Defect

Firstenberg¹,

Rousseau²

2012

Front Lines of Thoracic Surgery

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Front Lines of Thoracic Surgery 30 weeks after their initial acute event. It was these experiences that set the foundation for the belief that operative management should be delayed as long as possible to allow for scarring of the necrotic myocardium to provide for a more stable repair. As experiences grew -in terms of the initial diagnosis and surgical management -early repair was advocated, particularly in patients who were stable before hemodynamic deterioration and subsequent end-organ failure. Clinical presentationThe incidence of PI-VSD has decreased considerably over the years with advances in myocardial reperfusion strategies. Historically, up to 5% of all myocardial infractions were associated with mechanical complications such free-wall rupture, papillary muscle rupture, and PI-VSD (Agnihotri, 2008). With current treatment algorithms that advocate early and aggressive attempts at revascularization of the acute ischemic myocardial -such as thrombolytic therapy, early percutanous interventions with coronary stenting (PCI), and, less frequently, emergent coronary artery bypass surgery (CABG) -the overall incidence has dropped significantly. Large multi-centers studies evaluating the pathophysiologies of acute myocardial infarctions have shown a current incidence of approximately 0.2% of all AMI. With delays in therapies, or late clinical presentation, and the resulting increase in myocardial damage, this incidence increases up to 2%. Despite the relatively low risk of developing a PI-VSD, it account for a disproportionally high risk of mortality. Over 5% of all early deaths after AMI are attributed directly to the pathophysiologic complications of PI-VSD (Poulsen, 2008). The timing of the development of a PI-VSD can be quite variable with the average time to clinical presentation is between 2 and 4 days, however presentation can be as few as a few hours after AMI or as long as several weeks. Patient risk factors include gender, with men at a greater risk than women (3:2 ratio), increasing age, and current smoking history. The mean age of presentation in GUSTO was 62.5 years and ranged from 44 to 81 years (Crenshaw, 2000).

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Surgical treatment of ventricular septal defect complicating acute myocardial infarction. Experience of a north Italian referral hospital

Cited by 52 publications

References 21 publications

Ventricular septal rupture following myocardial infarction: Clinical, haemodynamic, angiographic profile and long-term outcome

Ventricular septal rupture following myocardial infarction: Clinical, haemodynamic, angiographic profile and long-term outcome

Percutaneous Closure of Postinfarction Ventricular Septal Defect

Post Myocardial Infarction Ventricular Septal Defect

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