The authors present the case of a 48-year-old man admitted for acute onset of paraplegia in a patient suffering from backaches for 1 week. The rapidly progressive motor disturbances had been evolving for approximately 12 hours. The entire spinal MRI showed an epidural mass at T4-T6 associated with extensive lesions of spondylodiscitis and a T7-T8 vertebral body loss of height. A large six-level laminectomy was performed. A tumoral etiology couldn't be entirely excluded intraoperatively so that no fusion has been done at that time. The pathological exam revealed acute inflammatory lesions with no argument in favor of a tumoral process. Bacteriological exam of the pathological specimen and stools cultures were positive for Salmonella brandenburg.An episode of gastroenteritis after the ingestion of a pizza has been evoked. The antibiotic medication was prescribed for 12 weeks. Postoperative evolution was favorable with a possible march between bars 6 weeks after. The authors emphasize the pseudo-tumoral presentation in an immunocompetent patient, the lack of complications and the post-ingestion mechanism. 328 Open Journal of Modern Neurosurgery spondylodiscitis (SD) and/or spinal epidural abscess (SEA). Terms as pyogenic and hematogenous are often added with claimed intention to point out the non-specific infectious character and, respectively, the most frequently encountered modality of spreading. Also, the term spontaneous is mostly reserved for infections other than post-surgical. The presence or the absence of a spinal epidural abscess could bring out a spinal cord compression which constitutes a neurosurgical emergency. That's why, for more practical reasons, we should classify spinal infections in spondylodiscitis (or vertebral osteomyelitis) with or without spinal epidural abscess. SD accounts for 2% of bony infections [1]. SEA comprises for 0, 2-2 cases for 10,000 hospital admissions [2]. Age. SEA occurs in patients aged 30 -60 and two times more frequently in men [2] [3] [4] [5]. Elderly people are more susceptible to developing SD [6] [7]. Level. The lumbar spine is affected in 50% of cases with SD [8] [9] [10]. The thoracic level was reported in over 50% of cases with SEA, followed by lumbar, then cervical [8] [11], with a prevalence of midthoracic spine (T6-T8) [12]. SEA appears more frequently in the cervical spine (90%) upon certain authors [13], or lumbar spine in another series [4] but causes a neurological deficit in the thoracic spine (80%) [13]. Over 80% are posterior to the cord [11] [14]. The lumbar spine seems to be more affected by Salmonella infection [15] [16] [17] [18] [19]. Some authors claim that thoracic SD with Salmonella in immunocompetent patients is extremely rare [20].