Supraventricular tachycardia in 23 cats; comparison with 21 cats with atrial fibrillation (2004–2014)

Greet, Victoria; Sargent, Julia; Brannick, M.; Fuentes, Virginia Luis

doi:10.1016/j.jvc.2020.04.007

Cited by 6 publications

(6 citation statements)

References 38 publications

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“…12,13 In contrast, there is paucity of feline SVT studies, with descriptions limited to case reports 9,[13][14][15][16][17][18][19][20] and one retrospective study. 21 In the study, the most common presenting clinical sign of SVT was respiratory distress, and the diagnosis of CHF was associated with shorter survival times, in a univariate analysis. 21 However, our report suggests there may be a subgroup of cats where CHF is secondary to SVT, and can be treated by adequate SVT management.…”

Section: Discussionmentioning

confidence: 85%

“… 21 In the study, the most common presenting clinical sign of SVT was respiratory distress, and the diagnosis of CHF was associated with shorter survival times, in a univariate analysis. 21 However, our report suggests there may be a subgroup of cats where CHF is secondary to SVT, and can be treated by adequate SVT management. Indeed, the retrospective study documented that a small number of cats with SVT had structurally normal hearts.…”

Section: Discussionmentioning

confidence: 85%

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Flash pulmonary oedema associated with paroxysmal supraventricular tachycardia: report of two cases

Seddon¹,

Serra

Seo

2023

Journal of Feline Medicine and Surgery Open Reports

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Case summary We describe two cats that had episodic tachypnoea and increased respiratory effort during periods of paroxysmal supraventricular tachycardia (SVT). Thoracic radiographs at the time of clinical signs were consistent with cardiogenic pulmonary oedema. Echocardiography following stabilisation revealed a hypertrophic cardiomyopathy phenotype with normal left atrial size in both cats. The first cat was initially treated with diltiazem, but this did not reduce the frequency of the clinical episodes. Diltiazem was switched to atenolol and the cat remained well without further recurrence. At the time of writing, the cat was reported to be well, 3 years after the initial diagnosis of SVT. The second cat was first managed with diltiazem and was then transitioned to atenolol due to recurrent clinical episodes. The episodes were less frequent with atenolol but still present. Therefore, atenolol was changed to sotalol. The cat remained well on sotalol for 2 years with only one recurrent episode during a painful event. The patient then suffered a sudden cardiac death, 5 years after the initial diagnosis of SVT. Relevance and novel information To our knowledge, this is the first report that describes flash pulmonary oedema developing secondary to episodic paroxysmal SVT in cats. Despite the severity and speed of respiratory compromise, prognosis may be good with an adequate arrhythmia control.

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Section: Discussionmentioning

confidence: 85%

Section: Discussionmentioning

confidence: 85%

Flash pulmonary oedema associated with paroxysmal supraventricular tachycardia: report of two cases

Seddon¹,

Serra

Seo

2023

Journal of Feline Medicine and Surgery Open Reports

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“…This case documents the clinical management of a cat in cardiogenic shock secondary to a suspected AP-mediated SVT, presumably without underlying structural cardiac or systemic disease. In the veterinary literature, the reported cases of syncope in cats have been associated with prolonged periods of sinus arrest, 13 right ventricular outflow tract obstruction secondary to heart worm disease, 14 increases in intrathoracic and intra-abdominal pressure with subsequent reduced venous return during defecation, 15 paroxysmal AV block with ventricular standstill, 16 ventricular tachycardia, 17 arrhythmogenic right ventricular cardiomyopathy, 18 AF and sustained SVT, 19 and recurrent syncope associated with paroxysmal SVT in a Devon Rex cat diagnosed by implantable loop recorder. 7 An episode of 'tetanic seizure' possibly associated with paroxysmal atrial tachycardia has been described in a Persian cat; however, a confirmative association between syncope and tachycardia was not observed.…”

Section: Discussionmentioning

confidence: 99%

Cardioversion of supraventricular tachycardia in a cat with suspected Wolff–Parkinson–White syndrome in the absence of structural cardiac disease

Miralles‐Plaza¹,

McDonald²

2021

Vet Record Case Reports

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The objective of this publication is to provide the clinical presentation, electrocardiographic findings and therapy for management of suspected Wolff-Parkinson-White syndrome in a cat without evidence of underlying cardiomyopathy or systemic disease. A 2-year-old male neutered domestic shorthair cat was presented for continued investigations following recent transient loss of consciousness episodes. Electrocardiography showed a sustained supraventricular tachycardia, which was successfully converted to sinus rhythm with intravenous diltiazem administration. Ventricular pre-excitation was presumed during the electrocardiogram postconversion and oral diltiazem was instituted. Cardiac troponin I was measured, conversely to prior reports, which was significantly high on presentation and decreased after treatment. The new information provided by this case report is that it represents an original description of Wolff-Parkinson-White syndrome in a cat without any obvious underlying cardiomyopathy, and treatment protocol resulted in a successful outcome to date.

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“…Arrhythmias can occur due to myocardial diseases, such as dilated cardiomyopathy or vice versa. Sustained tachycardia can cause dilated cardiomyopathy (10,12). It is difficult to distinguish between these two situations.…”

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confidence: 99%

“…In accordance with this view, the increase in creatine kinase levels in the case confirms myocardial damage. Cardiac troponins are used as the gold standard in humans to determine myocardial damage (12).…”

mentioning

confidence: 99%

Non-taurine responsive dilated cardiomyopathy in 2 cats

Sevim

Çolakoğlu

2021

Ankara Üniversitesi Veteriner Fakültesi Dergisi

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Feline dilated cardiomyopathy (DCM) is a rare disease characterized by myocardial failure and systolic dysfunction. Taurine deficiency is the most important cause of DCM phenotype in cats. A 2 year-old Scottish fold and a 8 month-old domestic shorthair cat referred to hospital with anorexia, weakness and increased respiratory effort. Based on electrocardiographic, radiographic and echocardiographic findings, the diagnosis of dilated cardiomyopathy was done in both the cases. The cases were managed with pimobendan, furosemide, acetylsalicylic acid and taurine. The cats were more active and clinically healthy during 15 days follow-up. No remarkable changes were observed in follow up echocardiographies. The cases presented here reflect clinical signs, cardiological examination findings, diagnosis and management of idiopathic dilated cardiomyopathy in 2 cats.

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Supraventricular tachycardia in 23 cats; comparison with 21 cats with atrial fibrillation (2004–2014)

Cited by 6 publications

References 38 publications

Flash pulmonary oedema associated with paroxysmal supraventricular tachycardia: report of two cases

Flash pulmonary oedema associated with paroxysmal supraventricular tachycardia: report of two cases

Cardioversion of supraventricular tachycardia in a cat with suspected Wolff–Parkinson–White syndrome in the absence of structural cardiac disease

Non-taurine responsive dilated cardiomyopathy in 2 cats

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