Superior Mesenteric Artery Syndrome

Valdes, Alberto; Cárdenas, Oscar; Espinosa, Adriana; Villazón, Oscar; Valdes, Viridiana

doi:10.1016/j.jamcollsurg.2005.04.033

Cited by 16 publications

(19 citation statements)

References 3 publications

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“…The presentation of SMA syndrome can be an acute or chronic course, depending on the severity of the obstruction, generally characterized by intermittent non-specific symptoms such as epigastric fullness, postprandial bloating, bilious emesis, crampy mid-abdominal pain, and weight loss (4,7,10,11). Interestingly, patients with SMA syndrome often find relief of symptoms by assuming a left lateral decubitus, prone, or knees-to-chest positioning, as seen in Case 1.…”

Section: Discussionmentioning

confidence: 99%

Emergency Department Presentation of Superior Mesenteric Artery Syndrome: Two Cases in Marine Corps Recruits

Rudinsky

Matteucci

2012

The Journal of Emergency Medicine

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Section: Discussionmentioning

confidence: 99%

Emergency Department Presentation of Superior Mesenteric Artery Syndrome: Two Cases in Marine Corps Recruits

Rudinsky

Matteucci

2012

The Journal of Emergency Medicine

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“…In rare cases, the syndrome may be acute with rapid evolving upper intestinal ileus [34, 55]. Whereas patients that underwent scoliosis surgery often show symptoms within the first postoperative week [27, 42, 43] and burn patients after 2 weeks [31], other patients suffer from non-specific symptoms for months [10, 56] to years [1, 7, 8, 17]. …”

Section: Symptomsmentioning

confidence: 99%

Recalling Superior Mesenteric Artery Syndrome

Welsch

Büchler²,

Kienle³

2007

Dig Surg

336

474

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Background: Superior mesenteric artery syndrome is uncommon and characterized by postprandial epigastric pain, nausea, vomiting, anorexia and weight loss. The syndrome is caused by compression of the third part of the duodenum in the angle between the aorta and the superior mesenteric artery. This review updates etiology, epidemiology, diagnosis, treatment and outcome of the superior mesenteric artery syndrome. Methods: Review of the literature. Results: Frequently, predisposing medical conditions associated with catabolic states or rapid weight loss result in a decrease of the aortomesenteric angle and subsequent duodenal obstruction. External cast compression, anatomic variants and surgical alteration of the anatomy following spine surgery or ileoanal pouch anastomosis can also precipitate the syndrome. Once radiologic studies have established diagnosis, first-line treatment is usually conservative with jejunal or parenteral nutrition for restoration of the aortomesenteric fatty tissue. If conservative management fails, surgical options include open or laparoscopic duodenojejunostomy or duodenal mobilization and division of the ligament of Treitz. Conclusion: Superior mesenteric artery syndrome is clearly defined and frequently associated with a wide range of predisposing conditions and surgical procedures; clinicians have to consider this syndrome in such a setting. Larger studies are needed to better define the optimal treatment for this disease.

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“…Superior mesenteric artery (SMA) syndrome is a rare cause of small bowel obstruction [4]. The diagnosis is confirmed by the loss of an angle between the superior mesenteric artery and the abdominal aorta to less than 20 degrees [5].…”

Section: Discussionmentioning

confidence: 99%