Sun Ra

Myers, Aaron

doi:10.1093/acref/9780195301731.013.43535

Cited by 5 publications

(13 citation statements)

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“…Generation length estimated as 9 years ( COSEWIC 2003 ). F values pre-1993 estimated from Myers (2007) . F values post-1993 estimated from Trzcinski et al (2006) .…”

mentioning

confidence: 99%

“…Generation length estimated as 2 years ( Ricker 1981 ). F estimates were from Myers (2007) . Annual F estimates were only available for a subset of years (1970–75) in most cases.…”

mentioning

confidence: 99%

“…Generation length estimated as 3 years ( Ricker 1981 ). F estimates were from Myers (2007) . Silver hake: Rates of phenotypic change calculated from Showell et al (2003) , Fig.…”

mentioning

confidence: 99%

“…Generation length estimated as 16 years (average for all American plaice stocks; Busby et al 2007 ). F estimates are mean values from Myers (2007) .…”

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confidence: 99%

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SYNTHESIS: Life history change in commercially exploited fish stocks: an analysis of trends across studies

Sharpe

Hendry

2009

Evolutionary Applications

309

271

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Age and size at maturation have declined dramatically in many commercial fish stocks over the past few decades – changes that have been widely attributed to fishing pressure. We performed an analysis of such trends across multiple studies, to test for the consistency of life history changes under fishing, and for their association with the intensity of exploitation (fishing mortality rate). We analyzed 143 time series from 37 commercial fish stocks, the majority of which originated from the North Atlantic. Rates of phenotypic change were calculated for two traditional maturation indices (length and age at 50% maturity), as well as for probabilistic maturation reaction norms (PMRNs). We found that all three indices declined in heavily exploited populations, and at a rate that was strongly correlated with the intensity of fishing (for length at 50% maturity and PMRNs). These results support previous assertions that fishing pressure is playing a major role in the life history changes observed in commercial fish stocks. Rates of change were as strong for PMRNs as for age and size at 50% maturity, which is consistent with the hypothesis that fishing-induced phenotypic changes can sometimes have a genetic basis.

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“…Generation length estimated as 9 years ( COSEWIC 2003 ). F values pre-1993 estimated from Myers (2007) . F values post-1993 estimated from Trzcinski et al (2006) .…”

mentioning

confidence: 99%

“…Generation length estimated as 2 years ( Ricker 1981 ). F estimates were from Myers (2007) . Annual F estimates were only available for a subset of years (1970–75) in most cases.…”

mentioning

confidence: 99%

“…Generation length estimated as 3 years ( Ricker 1981 ). F estimates were from Myers (2007) . Silver hake: Rates of phenotypic change calculated from Showell et al (2003) , Fig.…”

mentioning

confidence: 99%

“…Generation length estimated as 16 years (average for all American plaice stocks; Busby et al 2007 ). F estimates are mean values from Myers (2007) .…”

mentioning

confidence: 99%

See 2 more Smart Citations

SYNTHESIS: Life history change in commercially exploited fish stocks: an analysis of trends across studies

Sharpe

Hendry

2009

Evolutionary Applications

309

271

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“…Our analysis did not replicate sex-SNP interaction results from a previous study on asthma in European American subjects in which the polymorphism rs2549003, which is located near interferon regulatory factor 1 (IRF1) on chromosome 5q31, displayed a significant sex-SNP interaction on asthma with male-specific effects. 22 In our data set no trend for interaction (P 5 .89) or asthma association in male subjects (P male 5 .39) could be observed for rs2548997, which was described to be in complete LD with rs2549003 in the same study.…”

Section: Discussionmentioning

confidence: 41%

Doublesex and mab-3 related transcription factor 1 (DMRT1) is a sex-specific genetic determinant of childhood-onset asthma and is expressed in testis and macrophages

Schieck

Schouten

Michel

et al. 2016

Journal of Allergy and Clinical Immunology

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Association of Genetic Risks With Autism Spectrum Disorder and Early Neurodevelopmental Delays Among Children Without Intellectual Disability

et al. 2020

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IMPORTANCE Autism spectrum disorder (ASD) is highly heritable, and modest contributions of common genetic variants to ASD have been reported. However, the association of genetic risks derived from common risk variants with ASD traits in children from the general population is not clear, and the association of these genetic risks with neurodevelopment in infants has not been well understood.OBJECTIVE To test whether a polygenic risk score (PRS) for ASD is associated with neurodevelopmental progress at age 18 months and ASD traits at age 6 years among children from the general population. DESIGN, SETTING, AND PARTICIPANTSIn this cohort study, 876 children in the Hamamatsu Birth Cohort for Mothers and Children in Hamamatsu, Japan, underwent testing for the association of an ASD PRS with neurodevelopmental progress and ASD traits. Data collection began in December 2007 and is ongoing. Data analysis was conducted from April to December 2019. MAIN OUTCOMES AND MEASURES Summary data from the largest genome-wide association study were used to generate ASD PRSs, and significance of thresholds was calculated for each outcome. The Autism Diagnostic Observation Schedule 2 was used to measure ASD traits at age 6 years, and the Mullen Scales of Early Learning was used to measure neurodevelopmental progress at age 18 months.

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Sun Ra

Cited by 5 publications

References 0 publications

SYNTHESIS: Life history change in commercially exploited fish stocks: an analysis of trends across studies

SYNTHESIS: Life history change in commercially exploited fish stocks: an analysis of trends across studies

Doublesex and mab-3 related transcription factor 1 (DMRT1) is a sex-specific genetic determinant of childhood-onset asthma and is expressed in testis and macrophages

Association of Genetic Risks With Autism Spectrum Disorder and Early Neurodevelopmental Delays Among Children Without Intellectual Disability

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