2008
DOI: 10.1016/j.jpedsurg.2008.02.005
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Subtotal laparoscopic splenectomy and esophagogastric devascularization for the thrombocytopenia because of portal cavernoma—case report

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Cited by 10 publications
(9 citation statements)
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“…Forty‐nine publications were selected for review of full text and three duplicate publications, one article undergone LPS for pigs not human, and eight articles that we could not get detailed data were excluded from our review. Forty‐four [5, 6, 11–52] with a total of 252 patients undergoing LPS or LSS met the criteria for analysis. These included four case‐matched comparative studies.…”
Section: Resultsmentioning
confidence: 99%
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“…Forty‐nine publications were selected for review of full text and three duplicate publications, one article undergone LPS for pigs not human, and eight articles that we could not get detailed data were excluded from our review. Forty‐four [5, 6, 11–52] with a total of 252 patients undergoing LPS or LSS met the criteria for analysis. These included four case‐matched comparative studies.…”
Section: Resultsmentioning
confidence: 99%
“…The most common indications in these series were splenic cystic lesions( n = 84) [5, 6, 12–30, 51, 52], followed by splenic hematological diseases ( n = 70) [5, 6, 21, 24, 31, 33–35], non‐cystic intraparenchymal lesions ( n = 59) [5, 6, 20–24, 36–44, 50, 52], spleen rupture ( n = 22) [11, 45], splenomegaly of unknown origin ( n = 9) [5], splenic abscess ( n = 3) [23, 42, 49], severe splenic pain due to ischemia provoked by vascular obstruction of the spleen ( n = 2) [47], and each for Gandy–Gamna bodies, Benign metaplasia, and undiagnosed splenic lesion [22, 48]. The most common surgical procedures performed in these series were four‐trocar laparoscopic splenectomy ( n = 117) [14, 16–18, 20, 22–24, 28, 30, 31, 35, 38, 39, 42, 43, 45, 47, 48, 50, 51], followed by three‐trocar laparoscopic splenectomy ( n = 53) [5, 15, 19, 21,...…”
Section: Resultsmentioning
confidence: 99%
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“…The occurrence of vascular malformation in children suggests that a congenital defect is often a contributing factor, such as prior umbilical cannulation and infection. Inherited and acquired prothrombotic disorders (e.g., latent myeloproliferative disorder, protein C or protein S deficiency, and antiphospholipid syndrome) and thrombotic stimuli, such as pregnancy or oral contraceptives, intra-abdominal infection, and surgical procedures, have been observed in adults, while repeated abdominal infections, sepsis, abdominal invasive procedures, trauma, and congenital anomalies, with or without a prothrombotic state, have been alleged to lead to CTPV in children ( 16 19 ). After the exclusion of the aforementioned causes, the etiology of EHPVO remains obscure in up to 50% of patients ( 20 ).…”
Section: Etiologymentioning
confidence: 99%
“…In case of multiple publications by the same surgical team, to prevent data overlap when different variables were compared within group in each publication, only the more informative comparison was considered. Three articles were excluded from our analysis: a case report of 1 child 12 (our study focused on adults 18–80 years old); another case report 13 that addressed only 1 postoperative complication, gastric perforation, which was also mentioned in another article, 14 ; and a third article that did not report LSD. 15 …”
Section: Methodsmentioning
confidence: 99%