Suberosis Respiratory Disease in Cork Workers

Ávila, Ramiro; Villar, T. G.

doi:10.1016/s0140-6736(68)91239-7

Cited by 45 publications

(8 citation statements)

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“…There have been some reports of hypersensitivity pneumonitis (HP) associated with an occupational exposure to mould-contaminated wood dusts containing Cryptostroma corticate [6,7]. Atternaria [8], Rhizopus., Paecilomyces and Aspergiltus [9][10][11], Penicittium spp. [11][12][13].…”

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confidence: 99%

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Hypersensitivity pneumonitis induced by Penicillium expansum in a home environment

Park

Jung

Kim

1994

Clin Experimental Allergy

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An episode of fever, cough, shortness of breath and leucocytosis developed in a 31-year-old atopic housewife from mould exposure in her home environment is evaluated. A chest radiograph revealed diffuse tiny nodular infiltrations in both whole lung fields. Spirometry revealed a severe restrictive type of ventilation impairment. Bronchoalveolar lavage (BAL) showed an increased lymphocyte count with reversed CD4+/CD8+ ratio and transbronchial lung biopsy showed markedly increased lymphocytic infiltration in alveolar septa. Fungal cultures in the air of her home were positive for Penicillium expansum and other fungi. Double immunodiffusion test with the patient's serum showed two precipitin bands to P. expansum antigens. Her symptoms, abnormal findings of radiograph, and spirometric abnormalities disappeared after 2 months' avoidance. The serum precipitin disappeared after 1 month's avoidance. This study indicates that the patient had hypersensitivity pneumonitis (HP) on exposure to P. expansum in her home environment.

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“…Atternaria [8], Rhizopus., Paecilomyces and Aspergiltus [9][10][11], Penicittium spp. [11][12][13]. In this paper, we report a case of HP from mould exposure (P. expansum) in the home environment of a 31-year-old woman.…”

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confidence: 99%

Hypersensitivity pneumonitis induced by Penicillium expansum in a home environment

Park

Jung

Kim

1994

Clin Experimental Allergy

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“…Confirmatory investigations conventionally depend on serological methods-particularly gel diffusion precipitin tests. [1][2][3][4][5][6][7][8][9] While such tests usually demonstrate the presence of precipitating antibody if the antigen in question is indeed responsible for the disease, this is not always the case.7 12 Conversely, relatively high proportions of subjects similarly exposed, but apparently unaffected, may also give positive serological results. [13][14][15][16][17] The antibody response may consequently reflect exposure more than disease.…”

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confidence: 99%

Positive "alveolar" responses to antigen inhalation provocation tests: their validity and recognition.

Hendrick¹,

Marshall²,

Faux³

et al. 1980

Thorax

105

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“…The cork antigens were used in concentrations of 40 mg/ml and fungal antigens of 50 mg/ml. Immunoelectrophoresis The original technique of Scheidegger (1955) with slight modifications already reported (Avila, 1965) were used. Skin sensitivity tests An intradermal injection in the forearm of 0-2 ml of a solution of 5 mg/ml of Coca's fluid was made.…”

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confidence: 99%