Subclinical Cardiovascular Presentation in Covid19: A Case Report

Abstract

The coronavirus disease 2019 , elicited by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, is a pandemic public health emergency of global concern. Other than the profound severe pulmonary damage, SARS-CoV-2 infection also leads to a series of cardiovascular abnormalities, including myocardial injury, myocarditis and pericarditis, arrhythmia and cardiac arrest, cardiomyopathy, heart failure, cardiogenic shock, and coagulation abnormalities. mechanisms have been postulated for COVID-19-associated cardiovascular damage including SARS-CoV-2 receptor angiotensin-converting enzyme 2 (ACE2) activation, cytokine storm, hypoxemia, stress and cardiotoxicity of antiviral drugs. In this paper we reported a patient with severe Cocid 19 pnumonia that had very high level of Troponin I without history of cardio vascular and other underlying diseases expect mild HTN with use of amlodipine. She had a ECG with old LBBB without significant ST-T bnormality and normal data in Trance thoracic echocardiography(TTE). We named this condition subclinical involvemet of cardiovascular system in Covid19 and our discussion is about reasons and origination of this Troponin elevation.