Subacute cutaneous lupus erythematosus presenting with generalized poikiloderma

Pramatarov, Kyrill; Vassileva, Snejina; Miteva, Ljubka

doi:10.1016/s0190-9622(00)90141-5

Cited by 24 publications

(9 citation statements)

References 12 publications

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“…A targetoid appearance reminiscent of erythema multiforme may be seen. Other, less common skin manifestations include pityriasiform lesions, 20 mouth ulcers, livedo reticularis, periungual telangiectasias, erythroderma, 21 blisters, 22 vitiligo‐like lesions, 23 widespread pruritic plaques, 24 generalized poikiloderma, 25 leukocytoclastic vasculitis 26 and a diffuse non‐scarring alopecia. 4 Anti‐Ro/SSA antibodies are seen in 50–60 per cent of patients.…”

Section: Subacute Cutaneous Lupus Erythematosus (Scle)mentioning

confidence: 99%

The cutaneous pathology of lupus erythematosus: a review

2001

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The presentation of lupus erythematosus (LE) ranges from a skin rash unaccompanied by extracutaneous stigmata to a rapidly progressive lethal multiorgan disease. The diagnosis and subclassification is traditionally based on the correlation of serological and clinical findings. The latter include a photoinduced skin rash, arthralgia, arthritis, fever, Raynaud’s phenomenon, anemia, leukopenia, serositis, nephritis and central nervous sysdtem disease. The conventional classification scheme includes systemic, subacute cutaneous and discoid LE. Recent advances in our understanding of the cutaneous histopathology which correlates with the traditional forms of LE, along with certain novel LE subtypes, are the focus of this review. In addition to the main subtypes of LE, we will discuss associated vasculopathic lesions and the contribution of immunofluorescence microscopy to the diagnosis of LE and related connective tissue disease syndromes. Consideration will be given to unusual variants of LE such as anti‐Ro/SSA‐positive systemic lupus erythematosus (SLE), bullous SLE, lymphomatoid LE, lupus erythematosus profundus, drug induced LE, linear cutaneous LE, chiblains LE and parvovirus B19‐associated LE.

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Section: Subacute Cutaneous Lupus Erythematosus (Scle)mentioning

confidence: 99%

The cutaneous pathology of lupus erythematosus: a review

2001

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“…1 Poikiloderma may be caused by congenital diseases, autoimmune processes, and malignancy. 2–5 Poikiloderma of Civatte is a specific variant that originally was thought to be entirely related to cumulative sunlight exposure that is exacerbated by application of fragrances to the neck, which increases photoallergic phenomena. 6–8 Poikiloderma of Civatte is frequently accompanied by other sunlight‐related skin disorders such as wrinkles, atrophy in neighboring skin, cutis rhomboidalis nuchae, yellow papules and plaques of the face, colloid milium, diffuse telangiectasis, diffuse erythema, diffuse brown pigmentation, ecchymoses, freckles, actinic lentigines, nevi, Favre–Racouchot syndrome, and actinic keratosis.…”

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confidence: 99%

Treatment of Poikiloderma of Civatte with an Intense Pulsed Light Source

Weiß

Goldman

Weiss

2000

Dermatologic Surgery

134

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“…Unusual presentations of SCLE have been reported. These include one case of toxic epidermal necrolysis-like lesions, 5 one case with erythroderma, 6 one case of generalized poikiloderma, 7 and one case with erythroderma and acral bullae 8 . The diagnosis of SCLE in our case was based on the combination of clinical findings, histopathology, and positive Ro ⁄ SS-A.…”

Section: (A) (B)mentioning

confidence: 99%