Study of Atrioventricular Conduction in Man Using Electrode Catheter Recordings of His Bundle Activity

Damato, Anthony N.; Lau, Sun H.; Helfant, Richard H.; Stein, Emanuel; Berkowitz, Walter D.; Cohen, Stafford I.

doi:10.1161/01.cir.39.3.287

Cited by 148 publications

(20 citation statements)

References 21 publications

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“…Progressive A-V nodal delay with normal His-Purkinje conduction has also been noted when the heart rate of human subjects was increased by single atrial pacing. 13 Similarly under these conditions, the localization of A-V conduction delay to the N-H interval has been reported. "…”

Section: Discussionmentioning

confidence: 71%

A Study of Atrioventricular Conduction in Man Using Premature Atrial Stimulation and His Bundle Recordings

et al. 1969

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SUMMARYIn 20 subjects the effects of controlled premature atrial stimulation on atrioventricular conduction was studied using an electrode catheter technic for recording electrical activity of specialized conducting fibers. recording the electrical activity of the specialized conduction system.10' 1 Methods Right heart catheterization was performed on 20 subjects in the post-absorptive, nonsedated state. All subjects were in normal sinus rhythm at the time of study. Using local anesthesia, a tripolar electrode catheter* was percutaneously introduced into the right femoral vein and fluoroscopically positioned across the tricuspid valve. This catheter was used to record A-V nodal, His bundle, and right bundle-branch potentials as described in previous communications.10' 11 The proximal terminals of the electrode catheter were plugged into a multichannel distribution switch boxt which permitted the selection of any two electrodes for bipolar recordings, Each channel of the distribution switch box was led into the AC input of an ECG preamplifier. The filter frequencies of the ECG preamplifier were set at 40

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Section: Discussionmentioning

confidence: 71%

A Study of Atrioventricular Conduction in Man Using Premature Atrial Stimulation and His Bundle Recordings

et al. 1969

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“…The control sinus cycle length (CL) and AV nodal (AH) and infranodal (HQ) conduction times were measured. 16 The mean disopyramide level at the conclusion of repeat electrophysiologic testing was 2.9 + 0.9 ug/ ml. Because the therapeutic range for disopyramide is considered to be 2.0-4.0 ,ug/ml, only two patients (nos.…”

Section: Methodsmentioning

confidence: 91%

Electrophysiologic effects of disopyramide in patients with atrioventricular nodal dysfunction.

Wilkinson¹,

Desai²,

Hollister³

et al. 1982

Circulation

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“…It is used experimentally to facilitate the induction of tachyarrhythmias during electrophysiologic studies because sympathetic stimulation increases sinus rate, shortens atrioventricular intranodal conduction time, decreases excitability threshold, and shortens the plateau of diastolic depolarization of specialized cells, thus favoring the appearance of tachyarrhythmias (Damato et al, 1969;Hoffman and Ccinefield, 1964).…”

Section: Discussionmentioning

confidence: 99%

Effects of atenolol in patients with reciprocating supraventricular tachycardia

Pimenta

Pereira

1986

Clinical Cardiology

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Summary:We studied 12 patients with crisis of paroxysmal reciprocating supraventricular tachycardia before and after intravenous injection of 5 mg of atenolol. The patients were then followed for periods ranging from 6 to 50 months (median 34 months). During this time, they received oral atenolol therapy, at 200 mg for the first two weeks, and 100 mg daily thereafter. Tachycardia was due to reciprocation within the atrioventricular node in 9 patients, and to pre-excitation in 3 patients. Atenolol slowed the sinus rate, prolonged the atrioventricular conduction time, and increased the atrial cycle length at which atrioventricular nodal Wenckebach phenomenon occurred. During the tachycardia, atenolol increased the tachycardia cycle length, due to prolongation of the intranodal atrioventricular conduction time. Of the 11 patients who were observed for the full period, 7 had no further episodes of arrhythmia. One patient (with left-sided pre-excitation) failed to respond to any antiarrhythmic medication, one patient remained free of symptoms for two years, but received an atrial pacemaker for control of the tachycardia at the end of this period. Two patients (one with dual atrioventricular nodal pathways, and one with concealed leftsided pre-excitation) await other treatment for their tachycardia, after remaining free of symptoms for one and two years, respectively. These findings suggest that atenolol is an effective beta blocker for use in controlling arrhythmias in patients with reciprocating supraventricular tachycardia, for use in once daily dosage, and is a medication largely free of side effects.

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Study of Atrioventricular Conduction in Man Using Electrode Catheter Recordings of His Bundle Activity

Cited by 148 publications

References 21 publications

A Study of Atrioventricular Conduction in Man Using Premature Atrial Stimulation and His Bundle Recordings

A Study of Atrioventricular Conduction in Man Using Premature Atrial Stimulation and His Bundle Recordings

Electrophysiologic effects of disopyramide in patients with atrioventricular nodal dysfunction.

Effects of atenolol in patients with reciprocating supraventricular tachycardia

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