Stucco Keratosis

Willoughby, Cole

doi:10.1001/archderm.1972.01620090031007

Cited by 15 publications

(3 citation statements)

References 1 publication

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“…Stucco keratoses are small, whitish, keratotic plaques that are easily lifted off the skin with a fingernail, are situated principally on the extremities, and occur in middle‐aged or elderly persons. Although the cause is unknown, the condition is associated with dry skin and actinic exposure 1,2 . Multiple minute digitate hyperkeratosis is a familial disorder of keratinization, characterized by tiny, flaccid, digitiform projections and larger, flat‐topped to dome‐shaped papules, located on the trunk and limbs 3 .…”

Section: Discussionmentioning

confidence: 99%

“…Although the cause is unknown, the condition is associated with dry skin and actinic exposure. 1,2 Multiple minute digitate hyperkeratosis is a familial disorder of keratinization, characterized by tiny, flaccid, digitiform projections and larger, flat-topped to dome-shaped papules, located on the trunk and limbs. 3 Minute aggregate keratosis is another genetic disorder of keratinization with numerous, small, hyperkeratotic aggregates of papules and spicules and flat-topped hyperkeratotic papules on the trunk.…”

Section: Discussionmentioning

confidence: 99%

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Localized hypopigmented keratoses

et al. 2002

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A 16‐year‐old girl presented with a 2‐year history of hypopigmented, hard papules involving the extensor surfaces of the forearms and lower legs. Initially, 2–3‐mm‐sized papules were present on the forearms; gradually they increased in number and spread to involve the lower legs. Her family history was unremarkable. There was a history of worsening in the summer. On examination, she was skin type 3, with multiple, round, hypopigmented, keratotic, flat‐topped papules located on the distal dorsal parts of the extremities, with a stuck‐on appearance (Fig. 1). The papules were unrelated to follicular orifices, symmetrically distributed, and could not be detached. The lesions were not itchy. Her face, trunk, palms, and soles were spared, and no mucosal lesions were present. The nails and hair were normal. 1 Numerous, hypopigmented, flat‐topped papules on the arm Histologic examination of a papule revealed orthokeratotic hyperkeratosis, mild acanthosis, mild papillomatosis, and vacuolization in basal cells. A mild lymphocytic inflammatory response was seen. There were normal amounts of melanin in the basal cell layer and no melanophages were present (Fig. 2). 2 Hyperorthokeratosis, mild acanthosis, and papillomatosis. A mild inflammatory infiltrate is present in the dermis (hematoxylin and eosin, × 100) Despite some slight differences, the clinical and histologic features appeared to be similar to cases previously described under the title “disseminated hypopigmented keratoses”. The patient was treated with 0.1% tretinoin gel and 10% salicylic acid ointment and marked improvement was observed after several weeks.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Localized hypopigmented keratoses

et al. 2002

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“…Stucco keratosis was first described by Kocsard and Ofner in 1966, 1 who noted a high incidence in elderly Australians with 77% in men and 33% in women. In the U.S.A 2 . and Europe 3 this condition is often seen in elderly men.…”

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confidence: 99%

Successful treatment of stucco keratosis with maxacalcitol

Teraki¹,

Sato²,

Izaki³

2006

British Journal of Dermatology

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10 min, air dried, and dampened in deionized water. Tissue was scraped off the slides, suspended in 50-100 lL of a solution containing 0AE1 mol L )1 Tris-HCl (pH 8AE0) and 1 lg lL )1 proteinase K, incubated at 55°C overnight, boiled for 10 min, and finally stored at )20°C until analysed. To amplify HSV DNA using PCR, a pair of primers (Research Genetics, Huntsville, AL, U.S.A.) defining a 92-bp segment of the DNA polymerase gene was used, as described by Cao et al. 10 PCR reactions were performed with AmpliTaq DNA polymerase (Applied Biosystems, Foster City, CA, U.S.A.) under the following conditions: denaturation at 94°C for 45 s, annealing at 63°C for 30 s, and elongation at 72°C for 30 s. After 40 cycles of amplification, the reaction mixture was electrophoresed on a 2AE5% agarose gel (FMC, Rockland, ME, U.S.A.) and stained with ethidium bromide to visualize the DNA. Southern blot hybridization was performed with an internal 32 P-labelled HSVspecific oligonucleotide probe and was evaluated on X-ray film.All 24 PLE specimens that we examined were negative for HSV DNA by PCR analysis and subsequent Southern blot hybridization (Table 1, Fig. 1). In contrast, 10 of 31 EM specimens (32%) were positive for HSV DNA. This rate of HSV positivity in EM cases was comparable with 35-72% positivity rates seen in previous studies. 1,4,5 Interestingly, Yokoi et al. 4 detected HSV DNA in four of seven specimens from postherpetic EM but in none of three specimens from photoinduced EM. However, our inability to find HSV DNA presenting in any PLE skin samples contradicts the hypothesis that a direct immune response to HSV in the skin is involved in the pathogenesis of PLE.

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Non‐Melanoma Skin Cancer and Other Epidermal Skin Tumours

Mackie¹,

Quinn²

2004

Rook's Textbook of Dermatology

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Stucco Keratosis

Cited by 15 publications

References 1 publication

Localized hypopigmented keratoses

Localized hypopigmented keratoses

Successful treatment of stucco keratosis with maxacalcitol

Non‐Melanoma Skin Cancer and Other Epidermal Skin Tumours

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