2014
DOI: 10.3109/13816810.2014.973044
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Strabismus Surgery in Congenital Fibrosis of the Extraocular Muscles: A Paradigm

Abstract: Although patients with CFEOM present significant strabismus surgical challenges because of EOM dysinnervation, fibrosis, and/or heterotopia, satisfactory alignment and improvement of the head posture can be attained in a significant proportion of patients using an individually tailored surgical approach.

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Cited by 14 publications
(12 citation statements)
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“…Synergistic divergence can occur as an isolated finding or in the setting of CFEOM 16. Oystreck and associates35 performed genetic sequencing for patients with synergistic divergence who did not meet the clinical criteria for CFEOM and showed no mutations in KIF21A, PHOX2A, HOXA1 or ROBO3.…”
Section: Discussionmentioning
confidence: 99%
“…Synergistic divergence can occur as an isolated finding or in the setting of CFEOM 16. Oystreck and associates35 performed genetic sequencing for patients with synergistic divergence who did not meet the clinical criteria for CFEOM and showed no mutations in KIF21A, PHOX2A, HOXA1 or ROBO3.…”
Section: Discussionmentioning
confidence: 99%
“…or recession of a lateral rectus, in contrast, has not been as powerful as suturing it to the lateral orbital periosteum in decreasing the exotropia in CFEOM. [ 8 ] Merely cutting the muscle free of its attachment to the globe is often unpredictable. The muscle may reattach to the globe and continue to exact some force on the globe.…”
Section: Discussionmentioning
confidence: 99%
“…The traction suture was often used for patients with > 35 PD hypertropia and removed at 1 week after surgery. An alignment for vertical strabismus postoperative was considered as excellent with ≤ 10 PD, good with ≤ 20 PD and poor with > 20 PD [ 12 ].…”
Section: Methodsmentioning
confidence: 99%
“…CFEOM is a congenital, non-progressive restrictive strabismus sub-classified as CFEOM1, CFEOM2 or CFEOM3 and may be accompanied by ptosis and aberrant innervation [1]. There have been numerous studies on the clinical features, subclassification, surgical treatments and underlying genetic etiology of CFEOMs [1][2][3][4][5][6][7][8][9][10][11][12][13]. Results from previous genetic and neuroimaging studies have established that CFEOM is neurogenic, resulting in fibrotic tissue or atrophy of extraocular muscles due to deficiencies in the innervation of these muscles [4][5][6].…”
Section: Introductionmentioning
confidence: 99%