Staphylococcus aureus Pericarditis Masquerading as Anterior Mediastinal Mass

Nwiloh, Jonathan; Egbe, Patrick; Tagoe, A; Weaver, Lynn W.

doi:10.1378/chest.118.6.1832

Cited by 11 publications

(6 citation statements)

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“…To date, there are three other cases reported where a pericardial abscess was presented with symptoms of RV obstruction 1‐3 . We believe this is the first case reported with either pathology combined with intracardiac thrombus.…”

Section: Commentmentioning

confidence: 66%

“…Pericardial abscesses are rare 1 . The differential diagnosis includes active/chronic pericarditis 2 especially in the face of coexisting rheumatoid arthritis. The pericardium is rarely the primary site of infection, but is more often secondarily infected by hematogenous spread, direct spread from neighboring structures, or after cardiac surgery.…”

Section: Commentmentioning

confidence: 99%

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Pericardial Abscess Mimicking Intracardiac Neoplasm Causing Right Heart Failure

Karagounis

Stefanou

Sarsam

2005

Journal of Cardiac Surgery

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Purulent pericarditis involves the whole pericardium and usually presents as an acute illness with high mortality without prompt diagnosis and treatment. Presentation as a mediastinal mass causing compression of the right ventricle (RV) and symptoms of heart failure in patients without previous cardiac surgery is very rare and only three cases have been reported up to now (English language medical literature). Clinicians should be aware of this unusual condition for prompt diagnosis and treatment to be instituted.

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Section: Commentmentioning

confidence: 66%

Section: Commentmentioning

confidence: 99%

Pericardial Abscess Mimicking Intracardiac Neoplasm Causing Right Heart Failure

Karagounis

Stefanou

Sarsam

2005

Journal of Cardiac Surgery

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confidence: 99%

Primary pericardial abscess caused by Staphylococcus aureus infection without a predisposing condition

Nakao

Higaki

Nakama

et al. 2019

Journal of Cardiology Cases

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Pericardial abscess (PA) is a rare but serious life-threatening illness. Since the advent of antibiotic therapy, the incidence of PA has decreased, while the etiologic agents have changed from predominantly pneumococcus and streptococcal species to staphylococcus, anaerobes, and fungus [1]. Nowadays, PA is observed as a secondary complication of immunosuppression in hosts, chronic inflammation, drug users, or following trauma. We report the case of a patient with primary PA induced by Staphylococcus aureus infection without bacteremia, which developed to constrictive pericarditis. Case reportA 75-year-old man presented to our hospital with a lowgrade fever and a one-month history of worsening dyspnea. His medical history was myelodysplastic syndrome (MDS) without excess blasts, indicating good prognosis (international prognostic scoring system: scored as very low risk). On physical examination, the blood pressure was 113/61 mmHg, the pulse rate 53 beats per minute, and the temperature 37.3 C. Laboratory tests revealed a white blood cell count of 10,300/ mL (neutrophils, 81%), hemoglobin 7.5 g/dL, C-reactive protein 17.84 mg/dL, creatine kinase 69 U/L, creatine kinase myocardial band 16 U/L, and troponin T 0.038 ng/mL, which indicated inflammation, infection, and refractory anemia of MDS, but no sign of myocardial injury. His chest radiograph showed cardiomegaly without congestion. A twelve-lead electrocardiogram showed normal sinus rhythm and ST elevation in leads I, II,

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“…El bacilo de Calmette-Guerin (BCG) es una cepa viva atenuada de Mycobaterium bovis (M. bovis), especie del grupo Mycobacterium tuberculosis complex (1,2). La administración de BCG intravesical es eficaz en el tratamiento del carcinoma superficial de vejiga (1)(2)(3)(4). El mecanismo antitumoral parece ser una reacción de hipersensibilidad que causa una intensa infiltración por linfocitos y macrófagos, la cual facilita la eliminación de las células tumorales (1,5,6).…”

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“…La administración de BCG intravesical es eficaz en el tratamiento del carcinoma superficial de vejiga (1)(2)(3)(4). El mecanismo antitumoral parece ser una reacción de hipersensibilidad que causa una intensa infiltración por linfocitos y macrófagos, la cual facilita la eliminación de las células tumorales (1,5,6). Aunque infrecuente, dicha terapia puede presentar efectos secundarios potencialmente letales.…”

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