Standards for analysis of ventricular late potentials using high-resolution or signal-averaged electrocardiography: A statement by a task force committee of the European Society of Cardiology, the American Heart Association, and the American College of Cardiology

Breithardt, Günter; Cain, Michael E.; El‐Sherif, Nabil; Flowers, Nancy C.; Hombach, Vinzenz; Janse, Michiel J.; Simson, Michael B.; Steinbeck, Gerhard

doi:10.1016/0735-1097(91)90822-q

Cited by 387 publications

(46 citation statements)

References 73 publications

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“…Three conventional SA–ECG time domain indices were recorded at a 40 Hz bandpass filter setting: the duration of the total filtered QRS (fQRS) complex, the duration of the low-amplitude signal (LAS) after the voltage decreased to less than 40 μV, and the root mean square (RMS) of the amplitude of signals in the last 40 ms of the fQRS complex. The SA–ECG was considered predictive of ventricular arrhythmia if the fQRS duration was 120 ms or greater20 or if any two of the following three criteria were met: fQRS duration greater than 114 ms, LAS greater than 38 ms, or RMS less than 20 μV at a 40 Hz filter setting 21. A 24-h Holter recording was obtained within 48 h of the SA–ECG.…”

Section: Methodsmentioning

confidence: 99%

Reperfusion haemorrhage as determined by cardiovascular MRI is a predictor of adverse left ventricular remodelling and markers of late arrhythmic risk

Mather

Fairbairn

Ball

et al. 2010

Heart

140

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Section: Methodsmentioning

confidence: 99%

Reperfusion haemorrhage as determined by cardiovascular MRI is a predictor of adverse left ventricular remodelling and markers of late arrhythmic risk

Mather

Fairbairn

Ball

et al. 2010

Heart

140

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“…It could be argued that the signal averaged ECG might have been affected by the presence of right bundle branch block in our patients and thus be an inaccurate diagnostic tool 19. However, other investigators have found that signal averaged ECG findings may be helpful in detecting high risk patients despite the presence of right bundle branch block 12…”

Section: Discussionmentioning

confidence: 89%

Echocardiographic and signal averaged ECG indices associated with non-sustained ventricular tachycardia after repair of tetralogy of Fallot

Brili

Aggeli²,

Gatzoulis³

et al. 2001

Heart

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Objective-To identify any possible association between diVerent readily available non-invasive indices and potential malignant ventricular arrhythmias in patients with repaired tetralogy of Fallot. Design-27 consecutive patients, mean (SD) age 27.3 (11.7) years, were studied 15.7 (6.7) years after corrective surgery for tetralogy of Fallot, using 12 lead ECG, 24 hour Holter recordings, signal averaged ECG, and echocardiography. The following variables were measured: standard QRS duration, filtered QRS duration (fltQRS), low amplitude signal duration, and root mean square voltage of the last 40 ms of the fltQRS (RMS-40), as well as right ventricular systolic pressure, right ventricular ejection fraction, and the ratio of the maximum short axis diameters of the right and left ventricles (RD:LD). Results-All patients had right bundle branch block, with a mean QRS duration of 137.1 (14.9) ms. There were no patients with sustained arrhythmia. Five patients had runs of non-sustained ventricular tachycardia (group A) and the other 22 patients did not (group B). Univariate analysis showed that fltQRS and RD:LD ratio were significantly associated with nonsustained ventricular tachycardia. In addition, a fltQRS > 148 ms, low amplitude signal > 32.5 ms, RMS-40 < 23 µV, and RD:LD ratio > 1.05 were cut oV points with a high sensitivity for detecting patients with non-sustained ventricular tachycardia. Conclusions-Abnormal signal averaged ECG and echocardiographic variables are associated with potentially malignant ventricular arrhythmias on the Holter recordings in asymptomatic patients with repaired tetralogy of Fallot. (Heart 2001;85:57-60)

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“…It was suggested that VLP originate from viable areas around acutely infarcted myocardium [31] or interspersed between fibrous tissues of an old MI [32]. In such conditions, VLP is favored by disturbed architecture of myocardial fibers caused by necrosis and/or fibrosis that ultimately resulted in slow and fragmented depolarization [33].…”

Section: Discussionmentioning

confidence: 99%

Ventricular late potential in cardiac syndrome X compared to coronary artery disease

Lutfi

2017

BMC Cardiovasc Disord

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BackgroundAlthough ventricular late potential (VLP) was extensively studied in risk stratification of myocardial infarction (MI) patients, comparable researches evaluating presence of VLP in MI-free coronary artery disease (CAD) and cardiac syndrome X (CSX) subjects are scarce. This study aimed to compare presence of VLP between CSX and CAD patients.MethodsSignal average ECG (SAECG) was performed to 49 patients with a history of typical cardiac pain before undergoing diagnostic coronary angiography (DCA) in Al-Shaab cardiac center, Khartoum, Sudan. QRS duration, duration of the terminal part of the QRS complex with amplitude less than 40 microvolts (LAS40) and the root mean square voltage of the terminal 40 milliseconds (RMS40) of the filtered QRS complex were identified for each patient. Presence of two or more of QRS duration > 120 ms, RMS40 > 38 ms and LAS40 < 20 μV was considered indicative of VLP. Associations between VLP and patients grouped according to DCA results were assessed using appropriate statistical tests.ResultsVLP was present in 11.11% (3.63%–24.66%) and 15.38% (2.66%–42.23%) of patients with CAD and CSX respectively. Presence of VLP was comparable in patients with CAD and CSX (OR = 0.69, 95% CI = 0.11–6.05, P = 0.692), even after controlling for the possible variations in gender, age, body mass index (BMI), hypertension and diabetes mellitus in the studied groups.ConclusionPresence of VLP is comparable among CSX and CAD patients.

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Standards for analysis of ventricular late potentials using high-resolution or signal-averaged electrocardiography: A statement by a task force committee of the European Society of Cardiology, the American Heart Association, and the American College of Cardiology

Cited by 387 publications

References 73 publications

Reperfusion haemorrhage as determined by cardiovascular MRI is a predictor of adverse left ventricular remodelling and markers of late arrhythmic risk

Reperfusion haemorrhage as determined by cardiovascular MRI is a predictor of adverse left ventricular remodelling and markers of late arrhythmic risk

Echocardiographic and signal averaged ECG indices associated with non-sustained ventricular tachycardia after repair of tetralogy of Fallot

Ventricular late potential in cardiac syndrome X compared to coronary artery disease

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