Spontaneous nystagmus in benign paroxysmal positional vertigo

Stefano, Alessandro De; Kulamarva, Gautham; Citraro, Leonardo; Neri, G; Croce, A

doi:10.1016/j.amjoto.2010.01.005

Cited by 34 publications

(34 citation statements)

References 15 publications

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“…Finally, the data where matched with our result in treating idiopathic BPPV [19,22] and with patients complaining a "cervicogenic vertigo" following a whiplash injury to evaluate the differences.…”

Section: Methodsmentioning

confidence: 99%

Benign paroxysmal positional vertigo following whiplash injury: a myth or a reality?

Dispenza

Stefano

Mathur

et al. 2011

American Journal of Otolaryngology

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Objective: The aim of the study was to evaluate the true incidence, diagnosis, and treatment of benign paroxysmal positional vertigo (BPPV) arising after whiplash injury and to distinguish this type of posttraumatic vertigo from other types of dizziness complained after trauma.Methods: This was a retrospective study comprising patients referred to our center after whiplash injury. The patients were evaluated with neurotologic examination including bedside and instrumental tests. A Dizziness Handicap Inventory evaluating the symptoms of patients was submitted before and after treatment and was evaluated. The BPPV patients were separately evaluated from those with cervicogenic vertigo, and a comparison between our data about idiopathic BPPV was done.Results: Eighteen patients of whiplash who had BPPV were evaluated. The mean age was 38.2 years. BPPV was the cause of vertigo in 33.9% of total whiplash patients. In 16 cases, the posterior semicircular canal was involved; the lateral semicircular canal was involved in 2 cases. The instrumental neurotologic assessment did not show any alteration of either vestibulospinal reflexes or dynamic ocular movements. Duration of symptoms before treatment ranged from 3 to 26 days. A total of 55.5% of patients had relief from their symptoms after first repositioning maneuver. The Dizziness Handicap Inventory score improved in all patients treated with repositioning maneuvers, but no difference emerged with idiopathic BPPV data. Conclusion: BPPV after whiplash injury could be unveiled with a simple bedside examination of peripheral vestibular system, and a treatment could be done in the same session. The diagnosis of posttraumatic BPPV is not different from the idiopathic form, but the treatment may require more maneuvers to achieve satisfactory results.

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Section: Methodsmentioning

confidence: 99%

Benign paroxysmal positional vertigo following whiplash injury: a myth or a reality?

Dispenza

Stefano

Mathur

et al. 2011

American Journal of Otolaryngology

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“…3,9,10 Although their etiologies differ, the treatments of Iand S-BPPV are similar, and are based on canalith repositioning manoeuvres (CRMs), carried out after diagnosis. 4,5 Typically, I-BPPV and S-BPPV respond very well to these treatments; Pollak affirms that the resolution of symptoms is obtained after one manoeuvre in 78% of BPPV patients 11 and this data is largely confirmed in the English literature. 1,[12][13][14][15] Unfortunately, according to the literature, a small but insidious percentage of paroxysmal positional vertigo appears to be intractable after CRM and not self-limiting.…”

Section: Idiopathic Secondary Bppv and Malignant Paroxysmal Positionmentioning

confidence: 86%

“…This feature is very useful for differentiating LSC BPPV from other forms of peripheral or central vertigo. 4 The management of LSC BPPV includes several manoeuvres; the most common is the '360u roll' by Epley. 12 It consists of a complete roll of the patient on the examination bed, from supine to supine, away from the affected ear.…”

Section: Lateral Semicircular Canal Involvementmentioning

confidence: 99%

“…[3][4][5] The clinical features of BPPV are dominated by canalar symptoms in the acute condition (brief and intense spells of vertigo after hyperextension or hyperflexion of the head, lying down or standing up from lying position) and macular symptoms after the acute condition (prolonged instability which persists even after that patient has recovered from canalar symptomatology). 6 The treatment of this apparently intractable disease is actually a physical treatment: performing manoeuvres on the patient to restore the normal condition of the inner ear.…”

Section: Introductionmentioning

confidence: 99%

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Understanding benign paroxysmal positional vertigo: the management of lateral canal involvement

Stefano

2013

Physical Therapy Reviews

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“…This is likely due to the natural inclination of lateral semicircular canals (SCC) with respect to the horizontal plane. The nystagmus typically stops after forward flexion of the head in the sitting position (about 30°) (De Stefano et al, 2011). The pathologic gaze-evoked nystagmus due to brainstem or cerebellar lesions can also be accompanied by signs of vestibular imbalance.…”

Section: The Examinationmentioning

confidence: 99%