Spontaneous and Traumatic Vitreous Hemorrhage

Dana, Mohamad-Reza; Werner, Mark; Viana, Marlos A. G.; Shapiro, Michael J.

doi:10.1016/s0161-6420(93)31472-7

Cited by 61 publications

(34 citation statements)

References 9 publications

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“…The relative prevalence of these and other underlying conditions has varied in previous reports. [1][2][3][4][5] Despite recent improvements in ophthalmologic examination techniques, evaluation of vitreoretinal diseases with V-HEMO often presents a diagnostic challenge even when using the standard methods of A-and B-scan ultrasound (U/S). We conducted a retrospective study of the underlying causes of dense V-HEMO.…”

Section: Introductionmentioning

confidence: 99%

Comparison between clinical and ultrasound findings in patients with vitreous hemorrhage

Rabinowitz

Yagev

Shoham

et al. 2004

Eye

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Purpose To ascertain the causes of vitreous hemorrhage and to determine the accuracy of ultrasound (U/S) in these cases, based on the degree of agreement between ultrasound and clinical findings. Methods A chart review of 96 consecutive patients (106 eyes) with dense vitreous hemorrhage who underwent A-and B-scan U/S by one examiner between June 1996 and June 1999. U/S records were evaluated to determine the presence and exact distribution of areas of retinal detachment and the presence of posterior vitreous detachment, retinal tear, intraocular foreign body, or choroidal detachment. Clinical information was obtained from the medical records after the vitreous hemorrhage was reabsorbed or following vitreous surgery. Clinical and U/S findings were compared. Falsepositive and False-negative rates for U/S were calculated based on clinical findings. Results In 37 eyes (35%) the vitreous hemorrhage was because of proliferative diabetic retinopathy and in 33 eyes (31%) because of ocular trauma. The false-positive rate for retinal detachment (retinal detachment by U/S without clinical confirmation) was 18.9% (seven of 37 eyes). Retinal tears were diagnosed and localized accurately in only four of nine eyes (44%). Conclusions The most common cause of vitreous hemorrhage was proliferative diabetic retinopathy, followed by ocular trauma. U/S correctly diagnosed all cases of retinal detachment, but less than 50% of retinal tears. A total of 18.9% of the eyes were falsely diagnosed as having retinal detachment. U/S is an effective diagnostic tool in patients with vitreous hemorrhage.

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Section: Introductionmentioning

confidence: 99%

Comparison between clinical and ultrasound findings in patients with vitreous hemorrhage

Rabinowitz

Yagev

Shoham

et al. 2004

Eye

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“…Proliferative diabetic retinopathy is the most common cause of the spontaneous vitreous hemorrhage (VH) [1][2][3][4][5][6][7][8]. The presumed mechanism of diabetic VH is bleeding from fragile neovascular vessels caused by the tractional effect of acute posterior vitreous detachment (PVD) [5,7,8].…”

Section: Introductionmentioning

confidence: 99%

The predictive value of echography in diabetic vitreous hemorrhage

et al. 2007

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“…Proliferative diabetic retinopathy (PDR) is the most frequently reported cause of spontaneous vitreous bleeds, accounting for approximately one-third of all such cases; other causes include vitreous detachment with or without retinal tear and/or retinal detachment, retinal vein occlusion, proliferative sickle retinopathy, agerelated macular degeneration, and subarachnoid haemorrhage (Terson syndrome) [1,[6][7][8]. Ocular trauma is the leading cause of vitreous haemorrhage in young patients, particularly among males [2,[6][7][8], and may be associated with retinal detachment or retinal tear [9].…”

Section: Introductionmentioning

confidence: 99%

Successful use of recombinant activated factor VII in the treatment of vitreous haemorrhage: a report of seven cases

Al-Ameri¹,

Baker

2005

Blood Coagulation &Amp; Fibrinolysis

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Vitreous haemorrhage poses a serious threat to vision if untreated. Therapeutic options remain scarce and surgical intervention to resolve persistent bleeding is associated with risks that may further compromise vision. We report the use of recombinant activated factor VII (rFVIIa) in seven patients (six men, one woman; age, 30-65 years) with vitreous haemorrhage and severe reduction in visual acuity caused by trauma (n = 4) or proliferative diabetic retinopathy (n = 3). Initial doses ranged from 60 to 140 microg/kg; most patients received maintenance therapy with 20-60 microg/kg for at least 3 days. One patient received rFVIIa treatment for only 24 h and suffered a re-bleed, controlled successfully with further rFVIIa therapy. Five patients responded well to rFVIIa treatment, with reduced symptoms and improvements in visual acuity. Late presentation several days after trauma or symptom onset may have contributed to poor outcomes in the two patients who failed to respond to rFVIIa therapy. No adverse events were observed. An initial dose of rFVIIa 60-140 microg/kg, followed by 20-60 microg/kg repeated at 8-h intervals for 3-5 days, appears to be an effective therapeutic option for vitreous haemorrhage. However, further studies of rFVIIa use in this indication are warranted.

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Spontaneous and Traumatic Vitreous Hemorrhage

Cited by 61 publications

References 9 publications

Comparison between clinical and ultrasound findings in patients with vitreous hemorrhage

Comparison between clinical and ultrasound findings in patients with vitreous hemorrhage

The predictive value of echography in diabetic vitreous hemorrhage

Successful use of recombinant activated factor VII in the treatment of vitreous haemorrhage: a report of seven cases

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