Splanchnic concentrations and postprandial release of visceral adipokines

Circulating lipocalin 2 is neither related to liver steatosis in patients with non-alcoholic fatty liver disease nor to residual liver function in cirrhosis

“…Resistin does not differ between systemic, hepatic and portal venous blood in liver cirrhosis [30]. Similarly, LCN2 is comparable in these blood compartments.…”

“…Chemerin and resistin are associated with inflammation [28,29] and were recently analyzed in the patients with liver cirrhosis [25,30]. Only resistin was positively associated with LCN2 (r = 0.449, p = 0.005, Fig.…”

Predictors of Serum Levels of High Sensitivity C-Reactive Protein and Systolic Blood Pressure in Overweight and Obese Nondiabetic Women in Tehran: A Cross-Sectional Study

“…41 Saturated free fatty acids (FFAs) thus released can induce toll-like receptor-4 (TLR-4) expression, an innate immune response receptor, which in turn activates nuclear factor-kB (NF-kB) in macrophages, leading to proinflammatory cytokines secretion. 42,43 We found a significant relationship between hsCRP levels and cholesterol, TG, and LDL-C. By controlling FM and WC, however, this association disappeared in part of TG and cholesterol and it decreased with LDL-C. Therefore, this relationship between lipid profile and hsCRP levels might be secondary to the variation in fat mass and WC.…”

Effects of chronic ethanol consumption on levels of adipokines in visceral adipose tissues and sera of rats

“…Ethanol-induced liver injury may partly contribute to the increased insulin resistance index. Wiest et al [24] demonstrated that leptin, adiponectin, resistin and visfatin did not undergo hepatic clearance. They also observed similar leptin and adiponectin levels in patients with and without liver cirrhosis, while systemic visfatin levels were decreased and resistin levels were increased in liver cirrhosis [24] .…”