Spinal Cord Injury Following Osteoporotic Vertebral Fracture

O'Connor, Philip A.; Eustace, Stephen; O’Byrne, J.

doi:10.1097/00007632-200209150-00024

Cited by 19 publications

(8 citation statements)

References 12 publications

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“…The cases reported earlier have either single or adjacent vertebral fractures (19); however, the present case shows multiple non-adjacent fractures. According to the literature, neurological complications with vertebral compression fractures occur with less incidence, with gradual onset (20) and spontaneous recovery can occur (18). However, in our case, neurological impairment was developed only a week after the ominous back pain, which shows the rapidly progressive nature of the neurological damage.…”

Section: DI Is Sc Cu Us Ss Si Io On Nmentioning

confidence: 55%

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Asthmatic with Oral Sterid Usage Presenting with Multiple Fragility Fractures of Spine and Resultant Paraplegia: A Case Report

Riaz¹,

Jawa²

2017

Turk J Endocrinol Metab

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Section: DI Is Sc Cu Us Ss Si Io On Nmentioning

confidence: 55%

“…There have been published several reports and series on senile osteoporosis with spinal cord compression following minor or major trauma. Taggard and Tweedie (18). An 88-year-old lady, who was on oral steroids for 40 years, suffered a spontaneous fracture of T5 and gradual neurological impairment due to cord compression.…”

Section: DI Is Sc Cu Us Ss Si Io On Nmentioning

confidence: 99%

Asthmatic with Oral Sterid Usage Presenting with Multiple Fragility Fractures of Spine and Resultant Paraplegia: A Case Report

Riaz¹,

Jawa²

2017

Turk J Endocrinol Metab

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“…1). The type of OVF that can cause neurocompression results from either acute crush fracture [77,98,102] (Fig. 4) or delayed collapse of an antecedent wedge fracture that leads to retropulsion of a vertebral body fragment and contribution to progressive kyphotic deformity [71,75,97].…”

Section: Discussionmentioning

confidence: 99%

“…Neurological deficit can range from acute paraplegia (usually after an acute crush fracture) [98,102] to delayed onset of insidious paralysis that gradually deteriorates to severe paraplegia [69,73]. The latter phenomenon is usually associated with delayed vertebral collapse and progressive kyphotic deformity [75].…”

Section: Discussionmentioning

confidence: 99%

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Principles of management of osteometabolic disorders affecting the aging spine

Hadjipavlou

Katonis

Tzermiadianos

et al. 2003

European Spine Journal

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Osteoporosis is the most common contributing factor of spinal fractures, which characteristically are not generally known to produce spinal cord compression symptoms. Recently, an increasing number of medical reports have implicated osteoporotic fractures as a cause of serious neurological deficit and painful disabling spinal deformities. This has been corroborated by the present authors as well. These complications are only amenable to surgical management, requiring instrumentation. Instrumenting an osteoporotic spine, although a challenging task, can be accomplished if certain guidelines for surgical techniques are respected. Neurological deficits respond equally well to an anterior or posterior decompression, provided this is coupled with multisegmental fixation of the construct. With the steady increase in the elderly population, it is anticipated that the spine surgeon will face serious complications of osteoporotic spines more frequently. With regard to surgery, however, excellent correction of deformities can be achieved, by combining anterior and posterior approaches. Paget's disease of bone (PD) is a non-hormonal osteometabolic disorder and the spine is the second most commonly affected site. About one-third of patients with spinal involvement exhibit symptoms of clinical stenosis. In only 12-24% of patients with PD of the spine is back pain attributed solely to PD, while in the majority of patients, back pain is either arthritic in nature or a combination of a pagetic process and coexisting arthritis. In this context, one must be certain before attributing low back pain to PD exclusively, and antipagetic medical treatment alone may be ineffective. Neural element dysfunction may be attributed to compressive myelopathy by pagetic bone overgrowth, pagetic intraspinal soft tissue overgrowth, ossification of epidural fat, platybasia, spontaneous bleeding, sarcomatous degeneration and vertebral fracture or subluxation. Neural dysfunction can also result from spinal ischemia when blood is diverted by the so-called "arterial steal syndrome". Because the effectiveness of pharmacologic treatment for pagetic spinal stenosis has been clearly demonstrated, surgical decompression should only be instituted after failure of antipagetic medical treatment. Surgery is indicated as a primary treatment when neural compression is secondary to pathologic fractures, dislocations, spontaneous epidural hematoma, syringomyelia, platybasia, or sarcomatous transformation. Five classes of drugs are available for the treatment of PD. Bisphosphonates are the most popular antipagetic drug and several forms have been investigated. Keywords

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Klinik und Mortalitätsrisiko der osteoporotischen Wirbelfraktur

Becker

Ogon

Ballonkyphoplastie

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Spinal Cord Injury Following Osteoporotic Vertebral Fracture

Abstract: With increasing awareness of the morbidity attached to the osteoporotic spine, investigation and pharmacologic treatment are warranted and may reduce the possibility of catastrophic neurologic impairment as occurred in this case.

Cited by 19 publications

References 12 publications

Asthmatic with Oral Sterid Usage Presenting with Multiple Fragility Fractures of Spine and Resultant Paraplegia: A Case Report

Asthmatic with Oral Sterid Usage Presenting with Multiple Fragility Fractures of Spine and Resultant Paraplegia: A Case Report

Principles of management of osteometabolic disorders affecting the aging spine

Klinik und Mortalitätsrisiko der osteoporotischen Wirbelfraktur

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