Spectrophotometric determination of magnesium in biological fluids with xylidyl blue II

Chromý, V.; Svoboda, Vít; Степанова, Ю. А.

doi:10.1016/0006-2944(73)90075-6

Cited by 24 publications

(10 citation statements)

References 11 publications

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“…The volume of urine was measured to the nearest milliliter. Urine samples were analyzed for calcium by the o-cresolphthalein method [9], magnesium by the xylidyl blue method [10], phosphate by the molybdate method [11], uric acid by the uricase method [12], creatinine by the Jaffe reaction [13], and oxalate by the chromotropic acid reaction [14].…”

Section: Methodsmentioning

confidence: 99%

Urinary excretion of minerals, oxalate, and uric acid in north Indian children

Sweid

Bagga

Vaswani

et al. 1997

Pediatric Nephrology

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Urinary excretion of calcium, magnesium, phosphate, uric acid, oxalate, and creatinine was measured in 208 children (aged 8 -15 years, 124 boys, 84 girls), living in a residential school near New Delhi. Levels were reduced compared with those reported from developed countries. The 95th percentile value of 24-h creatinine excretion was 33.4 mg/kg, calcium 2.2 mg/kg, magnesium 2.9 mg/kg, phosphate 9.4 mg/kg, uric acid 4.4 mg/kg, and oxalate 1.5 mg/kg. The 95th percentile value of the urine calcium/creatinine ratio was 0.15 and oxalate/creatinine 0.06. The dietary intake of proteins, calcium, and other nutrients in these children was less than recommended and explained the reduced urinary excretion observed. Physicians need to be aware of the regional patterns of normal urinary excretion of these constituents.

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Section: Methodsmentioning

confidence: 99%

Urinary excretion of minerals, oxalate, and uric acid in north Indian children

Sweid

Bagga

Vaswani

et al. 1997

Pediatric Nephrology

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“…Serum Na ϩ , K ϩ and Cl Ϫ levels were measured using an ion selective electrode. Serum Ca 2ϩ and Mg 2ϩ levels were determined using O-CPC 16) and Xylidyl Blue II 17) methods, respectively. The activity of serum angiotensin converting enzyme (ACE) was assayed by Kasahara's method.…”

Section: Deep-sea Water Improves Cardiovascular Hemodynamics In Kurosmentioning

confidence: 99%

Deep-Sea Water Improves Cardiovascular Hemodynamics in Kurosawa and Kusanagi-Hypercholesterolemic (KHC) Rabbits

Katsuda

Yasukawa

Nakagawa

et al. 2008

Biological & Pharmaceutical Bulletin

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Water is essential to life processes and has a number of significant functions. The ingredients of drinking water as well as nutrients in foodstuffs are now considered important to people's health. Deep-sea water in particular has started to receive attention for its rich inorganic nutrients such as Mg, Ca, and K 1,2) which are due mainly to less photosynthesis of plant plankton and much organic decomposition. In addition to the beneficial effects of these minerals on the cardiovascular system, unknown effects of some ultratrace elements or unknown substances in deep-sea water may be found in future. Some scientific evidences of therapeutic or preventive effects of deep-sea water have been reported recently. Deepsea water improved mineral imbalances and atopic eczema/ dermatitis syndrome in humans 2) and was effective in the prevention of hyperlipidemia and atherosclerosis in cholesterol-fed rabbits. 3,4) We expect that the continuous intake of deep-sea water could prevent hypertension as well as hypercholesterolemia, because investigators have revealed that oral supplementation of calcium 5,6) or magnesium 7,8) to hypertensive patients lowered blood pressure in addition to reducing the serum total cholesterol level. 7)Hypercholesterolemia and hypertension occur due to genetic as well as dietary factors. The Kurosawa and KusanagiHypercholesterolemic (KHC) rabbit is an animal model of spontaneous hypercholesterolemia (Type IIa) and atherosclerosis established by inbreeding a mutant of the Japanese White rabbit discovered by Japan Laboratory Animals, Inc. in 1985.9) The KHC rabbit is deficient in LDL-receptors in the liver and shows an abnormally increased LDL-cholesterol level without unusual deposits of fat in some organs.9) Atherosclerosis develops in the aortic arch and around bifurcations of the main branch arteries by the age of 3 months.9) We also reported previously that young adult KHC rabbits aged 10-12 months showed mild hypertension compared to agematched normal rabbits in addition to hypercholesterolemia, though atherosclerotic lesions were relatively in the earlystage. 10,11) The mild hypertension is thought to be independent of hypercholesterolemia.11) Intervention of either hypercholesterolemia or hypertension is considered to be inadequate to prevent progression of atherosclerosis and related cardiovascular events if hypertension and hypercholesterolemia coexist.12) Ca and Mg in deep-sea water could suppress absorption of cholesterol in the small intestine in cholesterol-fed rabbits, which might contribute to anti-hypercholesterolemic action of deep-sea water.3,4) Therefore, it is appropriate to use spontaneous hypercholesterolemic rabbits.In the present study, we investigated the effect of the intake of refined deep-sea water at a degree of hardness of 1000 for 6 months on hypercholesterolemia and mild hypertension in KHC rabbits aged 4 months, at which time atherosclerosis starts to occur in the ascending aorta and around orifices of branch arteries, by measuring serum and plasma biochemical par...

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“…Blood Ca 2ϩ and Mg 2ϩ concentrations were measured immediately with ion-selective, flow-through, liquid-membrane electrodes on an automated electrolyte analyzer (AVL 988 -4 Electrolyte Analyzer; AVL Austria). (28) Total serum Ca and Mg were assayed by the cresolphthalein complexone method (29) and the xylidyl blue method, (30) …”

Section: Methodsmentioning

confidence: 99%

Bone Loss in Patients with Untreated Chronic Obstructive Pulmonary Disease Is Mediated by an Increase in Bone Resorption Associated with Hypercapnia

Dimai

Domej

Leb

et al. 2001

Journal of Bone and Mineral Research

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This study sought to determine whether the bone loss in untreated chronic obstructive pulmonary disease (COPD) is associated with hypercapnia and/or respiratory acidosis. Bone mineral density (BMD) measured at the distal forearm of the nondominant arm (with peripheral quantitative computed tomography [pQCT]) and serum markers of bone turnover were determined in 71 male patients with untreated COPD and 40 healthy male subjects who matched the patients in age, weight, and body mass index (BMI). The COPD patients, compared with controls, had reduced pulmonary functions, lower arterial pH, and elevated arterial partial pressure of CO 2 (P CO 2 ). The BMD (in T score) was significantly lower in COPD patients than that in control subjects (؊1.628 ؎ 0.168 vs. ؊0.058 ؎ 0.157; p < 0.001). The BMD of COPD patients correlated positively with arterial pH (r ‫؍‬ 0.582; p < 0.001), negatively with P CO 2 (r ‫؍‬ ؊0.442; p < 0.001), and negatively with serum cross-linked telopeptide of type I collagen (ICTP), a bone resorption marker (r ‫؍‬ ؊0.444; p < 0.001) but not with serum osteocalcin, a bone formation marker. Serum ICTP, but not osteocalcin, correlated with P CO 2 (r ‫؍‬ 0.593; p < 0.001) and arterial pH (r ‫؍‬ ؊0.415; p < 0.001). To assess the role of hypercapnia, COPD patients were divided into the hypercapnic (P CO 2 > 45 mm Hg; n ‫؍‬ 35) and eucapnic (P CO 2 ‫؍‬ 35-45 mm Hg) group (n ‫؍‬ 36). Patients with hypercapnia had lower BMD, lower arterial pH, and higher serum ICTP than did patients with eucapnia. Arterial pH and serum ICTP of eucapnic patients were not different from those of controls. To evaluate the role of uncompensated respiratory acidosis, COPD patients with hypercapnia were subdivided into those with compensatory respiratory acidosis (pH > 7.35; n ‫؍‬ 20) and those with uncompensated respiratory acidosis (pH < 7.35; n ‫؍‬ 15). The BMD and serum ICTP were not different among the two subgroups. In conclusion, this study presents the first associative evidence that the bone loss in COPD is at least in part attributed to an increased bone resorption that is associated primarily with hypercapnia rather than uncompensated respiratory acidosis. (J Bone Miner Res 2001;16:2132-2141)

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Spectrophotometric determination of magnesium in biological fluids with xylidyl blue II

Cited by 24 publications

References 11 publications

Urinary excretion of minerals, oxalate, and uric acid in north Indian children

Urinary excretion of minerals, oxalate, and uric acid in north Indian children

Deep-Sea Water Improves Cardiovascular Hemodynamics in Kurosawa and Kusanagi-Hypercholesterolemic (KHC) Rabbits

Bone Loss in Patients with Untreated Chronic Obstructive Pulmonary Disease Is Mediated by an Increase in Bone Resorption Associated with Hypercapnia

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