Spectral increment thresholds on a white background in different age groups of normal subjects and in acquired ocular diseases

Verriest, G; Uvijls, A

doi:10.1007/bf01569198

Cited by 28 publications

(10 citation statements)

References 19 publications

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“…Using traditional perimetric equipment the sensitivity of peripheral color thresholds had been fully described (Carlow, Flynn and Shipley, 1976;Verriest and Uvijls, 1977). Apparently on the assumption that adaptation is an important variable, the TiJbinger and Goldmann perimeters were modified to approach photopic adaptation levels (10 cd x m -2, Verriest and Uvijls, 1977;Israel and Verriest, 1977;Carlow, Flynn and Shipley, 1976) but few have attained rod saturation.…”

Section: Discussionmentioning

confidence: 99%

Results from a simple laser color perimeter

1984

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Section: Discussionmentioning

confidence: 99%

Results from a simple laser color perimeter

1984

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“…In this context, acquired colour vision losses are common 2 and can manifest characteristics similar to congenital defects. [2][3][4][5][6] Moreover, although many aspects of visual function can be affected by eye disease, colour vision changes often occur before the manifestation of other functional losses, such as alterations in visual acuity or visual fields. Colour vision is known to be affected in diabetes, [7][8][9][10][11][12] age-related maculopathy, [13][14][15][16] retinitis pigmentosa, 12 glaucoma, [17][18][19][20][21] optic neuropathies 22 and many other ocular diseases.…”

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confidence: 99%

“…4,16,22,[24][25][26][27][28] An altered perception of colour saturation can occur in one eye 4,5 or may be localised to one region of visual space thereby producing a relative decrease in the local saturation percept. [29][30][31] Colours are reported as less intense, appearing washed out and containing a greater amount of white, compared with adjacent unaffected regions or the fellow eye.…”

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Evidence for non‐selective colour channel involvement in diabetic eyes especially after laser treatment

Mahon

Vingrys

Fleiszig

1998

Clinical and Experimental Optometry

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Purpose:We consider the hypothesis that proliferative diabetes produces selective loss of colour channels. We also consider the possibility that laser treatment for this condition does not affect macula function. Methods: We tested for the possibility of a selective colour channel involvement in 35 eyes of 33 cases of proliferative diabetes by considering the outcomes of saturation and hue testing before and after pan-retinal photocoagulation (PRP). Saturation testing was achieved with the Sahlgren Saturation Test (SST) and hue testing was via the Farnsworth-Munsell 100 (FM100) hue test. Our results were compared to a recent model 1 that was developed to predict the saturation processing of diseased eyes. Results: All diabetic eyes with normal hue (FM100) scores passed the saturation test (SST). Most pre-treatment eyes (29 of 35 or 83 per cent) failed the FM100 hue test but only 16 of 35 (46 per cent) failed the saturation test. Following laser treatment, 97 per cent of eyes were abnormal on the FM100 hue test but only seven per cent failed the saturation test. Conclusion: Untreated diabetic eyes with proliferative retinopathy show losses of both hue and saturation processing. The nature of this relationship is consistent with that modelled assuming moderate and asymmetric non-selective losses in both chromatic and luminance channels. Laser treatment (PRP) appears to produce a paradoxical normalisation in saturation percepts in the presence of deteriorating hue scores. We find that this outcome is consistent with a more severe generalised reduction of sensitivity in both the chromatic and luminance processes and conclude that pan-retinal photocoagulation results in these unexpected macula changes. (Clin Exp Optom 1998; 81: 6: 272-279)

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“…We wanted to assess possible changes in the three color vision mechanisms and in their antagonism [Verriest and Uvijls, 1977], The background luminance used was 10 cd-nr2. The experimental conditions were the same as for color vision testing also concerning full pupil dilatation.…”

Section: Experiments With Assessment O F Color Vision Mechanismsmentioning

confidence: 99%

Modification by Fluoangiography of Color Vision in Diabetic Patients

Fontana¹,

Verriest²

1986

Ophthalmologica

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Fluoangiography causes in diabetics a temporary increase of the number of errors when reading Birch and SPP II pseudoisochromatic plates, especially in case of macular edema and for tritan plates. Other experiments show that this change is more due to an increased filter effect by the fluorescein accumulation than to the increase of glare by the flashes for photography. Measurements of the foveal spectral increment threshold sensitivity curves does not show impairment of the blue mechanism by fluoangiography; on the contrary the green and red mechanisms become less sensitive.

show abstract

Spectral increment thresholds on a white background in different age groups of normal subjects and in acquired ocular diseases

Cited by 28 publications

References 19 publications

Results from a simple laser color perimeter

Results from a simple laser color perimeter

Evidence for non‐selective colour channel involvement in diabetic eyes especially after laser treatment

Modification by Fluoangiography of Color Vision in Diabetic Patients

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