1948
DOI: 10.1111/j.1398-9995.1948.tb03302.x
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SOME VIEWS ON THE ASPIRIN‐HYPERSENSITIVE ALLERGY GROUP1

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Cited by 18 publications
(4 citation statements)
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“…Several investigations performed at this particular time (11,12,19) and subsequent studies have confirmed that aspirin-intolerant subjects with asthma have increased urinary excretion of cysteinyl-leukotrienes (20,21). On the basis of the well-established association between eosinophils and aspirin-intolerant asthma (22)(23)(24), and the indications that circulating eosinophils have an enhanced capacity to generate leukotrienes (25), the findings would seem to fit with the hypothesis that the eosinophils are one major source of the baseline production of leukotrienes.…”
Section: Leukotrienes As Mediators Of Persistent Airway Obstruction Imentioning
confidence: 83%
“…Several investigations performed at this particular time (11,12,19) and subsequent studies have confirmed that aspirin-intolerant subjects with asthma have increased urinary excretion of cysteinyl-leukotrienes (20,21). On the basis of the well-established association between eosinophils and aspirin-intolerant asthma (22)(23)(24), and the indications that circulating eosinophils have an enhanced capacity to generate leukotrienes (25), the findings would seem to fit with the hypothesis that the eosinophils are one major source of the baseline production of leukotrienes.…”
Section: Leukotrienes As Mediators Of Persistent Airway Obstruction Imentioning
confidence: 83%
“…A clinically salient and often early recognised feature of AIA is an increase in blood eosinophils [1,9]. This particular cell is known to have an efficient pathway for the biosynthesis of CysLTs [10], and we recently observed that activated sputum cells derived from subjects with AIA produced increased levels of CysLTs ex vivo that correlated with sputum eosinophil counts [11].…”
Section: Introductionmentioning
confidence: 97%
“…Salen and Arner 9 Walton and Randle 10 Giraldo et al 11 Falliers 12 Chafee & Settipane 7 Settipane et al 8 veyed, and the inclusion or exclusion of a pure nasal response without a decrease in pulmonary function. Based only on a history of adverse reactions to aspirin, Chafee and Settipane 1 found that the prevalence among allergic asthmatics was 4.3% and among those with allergic rhinitis, 0.7%.…”
Section: Investigatorsmentioning
confidence: 99%