“…Although the exact mechanism underlying this relationship has never been demonstrated, Ehrlich (1934) and Houston (1994a) suggested it involves N. faginata spores entering and germinating in beech phloem made accessible by C. fagisuga feeding wounds (Ehrlich, 1934;Houston, 1994a). Additionally, infection may be facilitated by C. fagisuga salivary enzymes that prevent trees from callusing wounded bark during heavy infestations (Parker, 1975;Lonsdale, 1980;Perrin, 1983;Houston, 1994a). The importance of particular phenols to disease development and resistance varies by host-pathogen system (Witzell and Martin, 2008).…”