Social cognitive interventions

Fiszdon, Joanna M.; Davidson, Charlie A.

doi:10.1016/b978-0-12-815315-4.00011-2

Cited by 4 publications

(2 citation statements)

References 73 publications

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“…"Blaming Bill" to exemplify when an individual has made an externalising-personalising attribution), which act as externalised scaffolds when generating alternative perspectives. Of at least 12 trials of SCIT, results show that emotion perception and ToM improve irrespective of clinical severity and setting (Fiszdon & Davidson, 2019). Externalising attributional biases to support perspective taking has also been adapted for use in other clinical populations, such as ASD (Turner-Brown, Perry, Dichter, Bodfish, & Penn, 2008) and in the SIFT IT protocol for people with brain injury described above (Cassel et al, 2020).…”

Section: Interventionmentioning

confidence: 99%

Remediating impairments in social cognition

Spikman¹,

Westerhof-Evers²,

Cassel³

2021

Clinical Disorders of Social Cognition

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Brain damage resulting from acute neurological disorders, such as traumatic brain injury (TBI) or stroke; insidious neurological disorders, such as brain tumors or multiple sclerosis (MS); or neurodegenerative diseases, such as frontotemporal dementia (FTD), can result in changes in social behaviour. This is particularly the case when prefrontal areas, or the circuits associated with these areas, are affected. Similar social behavioural presentations have been observed in various neurodevelopmental disorders, such as autism spectrum disorders (ASD), and neuropsychiatric disorders, such as schizophrenia spectrum disorders (SSD).In 1990, Neil Brooks, who extensively investigated sequelae of brain damage, remarked the following: "Of all three areas of deficit (physical, cognitive, behavioural) it is the behavioural deficit that lasts longest, is most difficult to treat, and has the most negative consequences on successful social and vocational rehabilitation" (p. 77). In this citation, he emphasised not only the disruptive nature of behavioural changes but also their immunity to treatment. Despite this, for a long time, clinical neuropsychology neglected social behavioural problems in brain disorders as a relevant domain. Proper assessment and treatment methods were sorely needed but yet to be developed. This changed with the emergence of the field of social neuroscience at the end of the previous century. Social neuroscientists acknowledged that humans have to survive in a complex social world; hence, brain anatomy and function have evolved in such a way so as to optimally facilitate social interactions (Brothers, 1990). We humans, thus, have a social brain with specialised areas dedicated to the processing of socially relevant information.Social cognitive information processing involves a series of stages (Adolphs, 2001;Beer & Ochsner, 2006). First, socially relevant information, e.g. expressions of emotions on other peoples' faces or in their voices (prosody), has to be attended to and perceived. Secondly, this perceived information, combined with knowledge regarding social rules and conventions, has to be interpreted, leading to an understanding of another's thoughts, feelings, and beliefs. In other words, this processing allows individuals to form a theory of other peoples' minds, and to take their perspective (the mentalising system; Frith & Frith,

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Section: Interventionmentioning

confidence: 99%

Remediating impairments in social cognition

Spikman¹,

Westerhof-Evers²,

Cassel³

2021

Clinical Disorders of Social Cognition

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“…Given the relationship between social cognition and functional outcome, a number of social cognitive interventions have been developed, ranging from single-domain, targeted treatments, to comprehensive treatments that aim to improve multiple domains of social cognition (8,9). In one of the first meta-analyses of social cognitive treatment trial data, these varied approaches to social cognitive treatments have collectively been associated with moderateto-large effects on emotion perception, psychiatric symptoms, and observer-rated functioning, with small-to-moderate effects on Theory of Mind (ToM), and no significant effects on attributional style (10).…”

Section: Introductionmentioning

confidence: 99%

Efficacy of social cognition and interaction training in outpatients with schizophrenia spectrum disorders: randomized controlled trial

Fiszdon,

Dixon,

Davidson

et al. 2023

Front. Psychiatry

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Given the relationship between social cognition and functional outcome in schizophrenia, a number of social cognitive interventions have been developed, including Social Cognition Interaction Training (SCIT), a group-based, comprehensive, manualized intervention. In the current trial, we examined SCIT efficacy as well as potential moderators of treatment effects. Fifty-one outpatients were randomized to SCIT or a wait-list-control (WLC), with assessments of social cognition, neurocognition, self-report, symptoms, and functioning conducted at baseline and end of the active phase. Relative to WLC, we did not find significant improvements for SCIT on neurocognition, social cognition, self-report, or symptoms, though there was a trend-level, medium effect favoring the SCIT condition on interpersonal and instrumental role function. Post-hoc analyses indicated that baseline neurocognition did not impact degree of social cognitive or functional change. Shorter duration of illness was significantly associated with better post-training neurocognition and self-esteem and, at trend-level with better symptoms and social functioning. We discuss the importance of outcome measure selection and the need for continued evaluation of potential treatment moderators in order to better match people to existing treatments.Clinical trial registration: Clinicaltrials.gov, Identifier NCT00587561.

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