Social Anxiety Disorder

Wong, Jillian W.; Gordon, Elizabeth A.; Heimberg, Richard G.

doi:10.1002/9781118156391.ebcp002027

Cited by 11 publications

(1 citation statement)

References 108 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Although there are well-validated treatments for SAD (Heimberg and Magee, 2014; Schneier et al, 2014; Wong et al, 2012), response rates for even the best empirically supported treatments suggest that many treated patients remain symptomatic. For instance, in one study, 35% of patients receiving the monoamine oxidase inhibitor phenelzine and 42% of patients receiving group cognitive behavioral therapy (CBT) were classified as non-responders (Heimberg et al, 1998).…”

Section: Introductionmentioning

confidence: 99%

Clinical presentation and pharmacotherapy response in social anxiety disorder: The effect of etiological beliefs

Cohen

Potter

Drabick

et al. 2015

Psychiatry Research

Self Cite

View full text Add to dashboard Cite

Therapies for social anxiety disorder (SAD) leave many patients symptomatic at the end of treatment and little is known about predictors of treatment response. This study investigated the predictive relationship of patients’ etiological attributions to initial clinical features and response to pharmacotherapy. Methods One hundred thirty-seven individuals seeking treatment for SAD received 12 weeks of open treatment with paroxetine. Participants completed the Attributions for the Etiology of Social Anxiety Scale at baseline in addition to measures of social anxiety and depression at baseline and over the course of treatment. Results A latent class analysis suggested four profiles of etiological beliefs about one’s SAD that may be characterized as: Familial Factors, Need to be Liked, Bad Social Experiences, and Diffuse Beliefs. Patients in the more psychosocially-driven classes, Need to be Liked and Bad Social Experiences, had the most severe social anxiety and depression at baseline. Patients in the Familial Factors class, who attributed their SAD to genetic, biological, and early life experiences, had the most rapid response to paroxetine. Conclusions These results highlight the effect of biological and genetically-oriented etiological beliefs on pharmacological intervention, have implications for person-specific treatment selection, and identify potential points of intervention to augment treatment response.

show abstract

Section: Introductionmentioning

confidence: 99%