Sino-atrial syncope

Easley, Robert M.; Goldstein, Sidney

doi:10.1016/0002-9343(71)90145-8

Cited by 71 publications

(11 citation statements)

References 38 publications

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“…Qualitative brief rhythm abnormalities often found in symptomatic patients include variant forms of the sick sinus syndrome [54][55][56][57], such as sinus pauses ( fig. 1) or the marked sinus overdrive suppression which may follow a brief burst of supraventricular tachycardia ( fig.…”

Section: Evaluation Of Symptomsmentioning

confidence: 99%

Clinical Applications of Ambulatory Electrocardiography

Kligfield

1984

Cardiology

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Section: Evaluation Of Symptomsmentioning

confidence: 99%

Clinical Applications of Ambulatory Electrocardiography

Kligfield

1984

Cardiology

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“…Encouraging results have been reported by some [9][10][11], suggesting that corticosteroids frequently re store sinus rhythm, particularly in acute complete A-V block [10][11][12]. Others, how ever, reported complete or almost complete failure [13,14], To the best of our knowledge, no such therapy has been reported in S-A block.…”

Section: Introductionmentioning

confidence: 77%

“…Although pharmacological treat ment of S-A disease has not been estab lished, several agents have been recently pro posed, including beta-adrenergic medica tions [4,5], amiodarone [6], theophylline [7] and hydralazine [8]. However, tachyarrhyth mias, frequently complicating the course of the bradycardic S-A disease, limit the useful ness of sinus node stimulators [9].…”

Section: Introductionmentioning

confidence: 99%

Corticosteroids in the Management of Sinoatrial Block

Abinader

Oliven²,

Kogan³

1988

Cardiology

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This report presented our experience in the treatment of sinoatrial (S-A) conduction disturbances with corticosteroids. Three patients with intermittent second-degree S-A block who failed to respond to atropine and isoproterenol were treated with prednisone for 6–17 weeks. Sustained improvement in S-A conduction following prednisone administration was confirmed by repeated Holter monitoring on and off therapy. Two patients eventually regained permanent sinus rhythm, while therapy led to marked diminution in S-A block in the 3rd patient. The possible mechanisms by which steroids may improve S-A conduction are discussed. Our observation is based on a small case series, each patient serving as his own control, and as such constitutes an indication for further confirmatory studies.

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“…This diagnosis is based on the presence of slow heart rate, sinoatrial block, paroxysmal atrial fibrillation and sup pression of the sinus node after spontaneous cessation of atrial fibrilla tion [2], According to the recent studies of H indm an et al [3] and D r e ifu s et al [1], the combination of sinus node dysfunction and the WPW syndrome is more than coincidental. H in dm an et al |3], using Holter monitoring, demonstrated a relatively high incidence of sinus node dysfunction in patients with WPW syndrome.…”

Section: Discussionmentioning

confidence: 99%