Síndrome poliúrico

F, Edda Lagomarsino; N, Ana Nardiello; Aglony, Marlene

doi:10.4067/s0370-41062004000300009

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“…Diabetes Mellitus) o no osmótica (DI). La causa más común es la DI central (DIC) o neurogénica producida por déficit de secreción de ADH 5 ; menos común es la DI nefrogénica (DIN) que se produce por una resistencia a la acción de la ADH a nivel de los túbulos colectores renales y, en último lugar, la polidipsia primaria también llamada polidipsia psicógena, donde se produce una inhibición de la secreción de ADH secundario a una ingesta excesiva de agua 6,7 .…”

Section: Introductionunclassified

Diabetes Insípida en Pediatría: Serie Clínica y Revisión de la Literatura

et al. 2009

Rev. chil. pediatr.

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Diabetes Insipidus in Pediatrics Introduction: Diabetes insipidus (DI) is a syndrome characterized by polyuria and polydipsia secondary to a decreased secretion or action of the antidiuretic hormone (ADH). An early diagnosis is essential. Diagnosis is made by measuring plasma and urinary osmolarity and their changes under water deprivation and after DDAVP administration. Objective: To describe the clinical, radiological characteristics as well as the initial treatment of eight children with DI, 3 of them nephrogenic DI (DIN) and 5 with central DI. Methods: A Retrospective, descriptive study in DI patients under control at the Catholic University of Chile and Sotero del Rio Hospital between 1998-2008 is presented. Clinical files were evaluated collecting clinical, epidemiologic, biochemical and image data. Serum (Sosm) and urinary osmolarity (Uosm) were registered. DI was diagnosed with a Sosm > 300 and Usm < 600 mOsm/L. Central DI was defined as the inability to reach a Uosm > 600 or a 50%-increase after DDAVP treatment. Otherwise DI was classified as DIN. Results: Eight patients (5 males) were studied. Chief complaints were polydipsia/polyuria (5/8), hyperthermia (2/8), and failure to grow (1/8). MRI showed endocraneal lesion in all patients with Central DI. All of these utilized oral or inhalatory DDAVP treatment. Patients with Nephrogenic DI were trated with Hydrochlrothiazide. Conclusion: Polydipsia, polyuria, hyperthermia with hypernatremia are suggestive of DI in the first year of life. Water deprivation test is diagnostic in differentiating Central and Nephrogenic DI. MRI is an essential diagnostic tool in CDI. Manegement should be multidisciplinary, including a pediatician, nephrologist, endocrinologist and nutricionist.

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Section: Introductionunclassified

Diabetes Insípida en Pediatría: Serie Clínica y Revisión de la Literatura

et al. 2009

Rev. chil. pediatr.

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Síndrome poliúrico

Cited by 1 publication

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Diabetes Insípida en Pediatría: Serie Clínica y Revisión de la Literatura

Diabetes Insípida en Pediatría: Serie Clínica y Revisión de la Literatura

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