Síndrome de Wellens: reconociendo el peligro

Carvajal, Carlos A.; Ardila, Diego J.

doi:10.1016/j.rccar.2015.04.002

Cited by 3 publications

(6 citation statements)

References 25 publications

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“…Therefore, it has been classified as unstable angina and is an occasional presentation of acute coronary syndrome. This clinic corresponds to the majority of cases reported in the medical literature [7][8][9]12]. The electrocardiogram reported negative T waves in DI and AvL, V1, V4 -V6 and biphasic in V2-V3, classifying as Wellens pattern type A or type 1, which corresponds to the minority of cases (24%) [7] and is the one with the highest sensitivity and specificity [8].…”

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confidence: 58%

“…In the literature consulted, several authors [5,8,12] state that the mechanisms associated with electrocardiographic changes are not entirely clear; but it has been proposed that they are related to a myocardium stunned by reperfusion, due to occlusion of the flow of the left anterior descending coronary artery that is spontaneously restored and it is believed that other elements apart from the coronary arteriosclerotic lesion can produce them, such as left ventricular edema, coronary spasm, right ventricular overload, microvascular disease, or nonspecific disorders of myocardial repolarization.…”

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confidence: 99%

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Wellens syndrome Type A. Case report

Correa,

Quintero Chang,

Candebat Correa

2023

JOJPH

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Wellens syndrome is an electrocardiographic manifestation of critical stenosis of the anterior left descending coronary artery, the persistent reversal of the T-wave in the V2 and V3 shunts. It was decided to report the case of a 48-year-old female patient with the aim of describing her clinical and electrocardiographic characteristics. The patient goes to the Emergency Room of Saturnino Lora Provincial Hospital referring oppressive precordial pain of 15 days of evolution, where an electrocardiogram and coronary angiography are performed, being diagnosed with Wellens Syndrome type A. It was considered to report this case because it is not an immediate recognition syndrome and the findings predict with a high sensitivity and specificity the compromise of the proximal part of the anterior descending artery, requiring an early invasive strategy with the aim of avoiding complications such as acute myocardial infarction and death.

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confidence: 58%

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confidence: 99%

Wellens syndrome Type A. Case report

Correa,

Quintero Chang,

Candebat Correa

2023

JOJPH

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“…Wellens syndrome was described in 1982 by Wellens et al, who characterized a subgroup (18%) of patients with unstable angina 2 ; discrete enzymatic changes; and 2 patterns that involved the T-wave in the precordial leads, with intermittent chest pain at the time of onset and with the high risk of developing an extensive anterior wall myocardial infarction because of the compromise of the LAD. 1 , 2 , 3 …”

Section: Discussionmentioning

confidence: 99%

“…In both patterns, there is adequate R-wave progression in the precordial leads, absence of ST-segment elevation or ST-segment depression, and no pathologic Q waves, and typically the evolution from one pattern to another is not described. 1 , 2 , 4 …”

Section: Discussionmentioning

confidence: 99%

Progression From Patterns A to B in Wellens Syndrome

Rodríguez,

Salva,

Aladio

et al. 2023

JACC: Case Reports

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“…Sin embargo en algunas ocasiones la CNG se presenta con arterias coronarias normales, un MINOCA como el caso que presentamos, en el que se ha podido producir una revascularización (45), aunque en la cronificación de este patrón no exista más que una isquemia epicárdica severa que produce la inversión de la repolarización en aVR como ocurre en nuestro caso .…”

Section: St -T Negativas En Cara Antero Lateral Lateral Alta E Inferunclassified

Is the apical hypertrophic cardiomyopathy the evolution of a myocardial infarction and non obstructive coronary arteries ( minoca )?

Fernández¹

2019

an. ranm

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La cardiopatía isquémica aguda se diagnostica en base a la clínica, con el dolor anginoso, los cambios electrocardiográficos (ECG) y las modificaciones en los niveles plasmáticos de los biomarcadores de daño miocárdico, principalmente troponina I (Tp) y creatínfosfokinasa (CPK). Las complicaciones eléctricas y mecánicas, se tratan de evitar con un manejo adecuado, basado en el rápido diagnóstico y la revascularización más temprana posible de la arteria ocluida o evitando la oclusión de las obstrucciones subtotales. Esta estrategia ha modificado la desfavorable evolución del Infarto Agudo de Miocardio (IAM), ha reducido drásticamente su mortalidad temprana y sus complicaciones tardías. Para que esto haya ocurrido han cambiado una serie de conceptos. En primer lugar, el mejor conocimiento de la secuencia de manifestaciones de los primeros momentos del infarto, ha obligado a cambiar al concepto de Síndrome Coronario Agudo (SCA). Presentamos el caso clínico de un Síndrome Coronario Agudo con Elevación del Segmento ST (SCACEST) con algunas características clínicas y ECG que podían hacer sospechar severidad. Discutimos las distintas formas de presentación y entidades responsables de los infartos de miocardio sin obstrucción de arterias coronarias,(MINOCA), y el porqué un SCACEST puede ser el inicio de la modificación del fenotipo de una Miocardiopatia Hipertrófica (MCH) no expresada hasta entonces y su transformación en una MCH apical.

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Síndrome de Wellens: reconociendo el peligro

Cited by 3 publications

References 25 publications

Wellens syndrome Type A. Case report

Wellens syndrome Type A. Case report

Progression From Patterns A to B in Wellens Syndrome

Is the apical hypertrophic cardiomyopathy the evolution of a myocardial infarction and non obstructive coronary arteries ( minoca )?

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