Síndrome de Ramsay-Hunt causante de parálisis laríngea

Gómez-Torres, Antonio; Vallejo, Antonio Medinilla; Jiménez, Antonio Abrante; Ortega, Francisco

doi:10.1016/j.otorri.2011.08.003

Cited by 5 publications

(5 citation statements)

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“…RHS is characterized by the involvement of the geniculate ganglion of the VII nerve and typically presents with a facial nerve palsy and vesicles in the ipsilateral external auditory canal. Although the VIII nerve is frequently co-involved, resulting in the development of dizziness and hearing loss, multiple lower cranial nerve involvement due to RHS has rarely been described [3] ; available reports in the literature include zoster laryngitis related to RHS with cranial polyneuropathy [4] , [5] , [6] , RHS involving the facial and superior laryngeal nerves in a patient with chronic renal disease [7] , RHS involving the VII, VIII and X cranial nerves in an immunocompetent individual [8] , severe dysphagia due to RHS involving the cranial nerves V, VII, VIII, X, and XII [9] , and RHS complicated by seven cranial nerve involvement (V, VII, VIII, IX, X, XI, and XII) [10] .…”

Section: Discussionmentioning

confidence: 99%

“…For instance, RHS without vesicles, condition known as zoster sine herpete [2] , is difficult to distinguish itself from Bell's palsy. Although the vestibulocochlear nerve is frequently co-involved during the course of RHS, multiple lower cranial nerve involvement has rarely been described in the literature [5] , [6] , [7] , [8] , [9] , [10] , [11] , [12] . Additionally, laryngitis due to VZV associated with RHS is not a well-recognized clinical entity.…”

Section: Introductionmentioning

confidence: 99%

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Ramsay Hunt syndrome and zoster laryngitis with multiple cranial nerve involvement

Shinha¹,

Krishna²

2015

IDCases

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Ramsay Hunt syndrome is characterized by varicella zoster virus infection affecting the geniculate ganglion of the facial nerve. It typically presents with vesicles in the external auditory canal associated with auricular pain and peripheral facial nerve paralysis. Although vestibulocochlear nerve is frequently co-involved during the course of Ramsay Hunt syndrome, multiple lower cranial nerve involvement has rarely been described in the literature. In addition, laryngitis due to varicella zoster virus is a diagnostic challenge due to its unfamiliarity among clinicians. We report a case of Ramsay Hunt syndrome with laryngitis involving multiple lower cranial nerves.

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Section: Discussionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Ramsay Hunt syndrome and zoster laryngitis with multiple cranial nerve involvement

Shinha¹,

Krishna²

2015

IDCases

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“…Dentro de las neuropatías encontramos a la neuritis periférica difusa, la cual puede comprometer a los pares craneales V, IX y XII (21,41,42). En el caso de las infecciones del sistema nervioso central (SNC), la encefalitis, asociada con el VVZ, se relaciona generalmente con la infección por el virus del herpes simple y puede presentar características clínicas adicionales a las descritas anteriormente como hemiparesia, hemihipoestesia o dismetría, y se presenta principalmente en huéspedes inmunosuprimidos, con cáncer o que usan esteroides (21).…”

Section: Complicacionesunclassified

Síndrome de Ramsay Hunt: revisión narrativa

Becerra-Mejía¹,

Roa-Gómez²,

Mendoza-Durán³

et al. 2021

Acta otorrinolaringol cir cabeza cuello

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El síndrome de Ramsay Hunt (SRH) corresponde a la asociación de la parálisis facial periférica con una erupción vesicular localizada en el pabellón auricular,causada por el compromiso del ganglio geniculado secundario a una infección por el virus de la varicela-zóster (VVZ). Este síndrome es la segunda causa máscomún de parálisis facial atraumática y representa aproximadamente el 10 %-12% de las parálisis faciales agudas, con una incidencia anual de 5 por cada 100 000 habitantes en Estados Unidos. El diagnóstico es principalmente clínico y entrelas manifestaciones más destacadas se encuentran síntomas neurológicos como otalgia, tinnitus, hipoacusia asociada con parálisis facial junto a lesiones herpéticas características. Dentro de las complicaciones que se pueden presentar en estaentidad se encuentra, principalmente, la neuralgia posherpética, seguida de otras menos frecuentes como la encefalitis, el herpes zóster oftálmico y la mielitis. El manejo actual del SRH se basa en la aplicación de terapias duales con corticosteroidesasociados a terapia antiviral, lo cual ha demostrado que el inicio temprano del tratamiento mejora el pronóstico y disminuye la aparición de complicaciones. El pronóstico de esta patología es inferior en comparación a patologías menos severas que comprometen el nervio facial (como la parálisis de Bell) y se ve impactado por varios factores como el inicio oportuno de tratamiento, el grupo etarioy la presencia de comorbilidades.

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“…[ 77 ] Clinically, it is characterized by 7 th cranial nerve palsy, otic pain, and herpetic vesicles around the auricle and external auditory canal. [ 77 78 ] Occasionally, LCN may be additionally involved, particularly the vagal nerve which may manifest with transient dysphagia. [ 77 78 ] Jugular-foramen-syndrome is characterized by acute onset dysphagia, dysphonia, and accompanied or preceded by cranial, cervical, or pharyngeal pain.…”

Section: Disorders Of Lower Cranial Nervesmentioning

confidence: 99%

“…[ 77 78 ] Occasionally, LCN may be additionally involved, particularly the vagal nerve which may manifest with transient dysphagia. [ 77 78 ] Jugular-foramen-syndrome is characterized by acute onset dysphagia, dysphonia, and accompanied or preceded by cranial, cervical, or pharyngeal pain. [ 13 ] The most frequent cause of Jugular-foramen-syndrome is herpes zoster oticus with herpetic eruptions of the skin or the mucosa.…”

Section: Disorders Of Lower Cranial Nervesmentioning

confidence: 99%

Disorders of the lower cranial nerves

Finsterer

Grisold

2015

Journal of Neurosciences in Rural Practice

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Lesions of the lower cranial nerves (LCN) are due to numerous causes, which need to be differentiated to optimize management and outcome. This review aims at summarizing and discussing diseases affecting LCN. Review of publications dealing with disorders of the LCN in humans. Affection of multiple LCN is much more frequent than the affection of a single LCN. LCN may be affected solely or together with more proximal cranial nerves, with central nervous system disease, or with nonneurological disorders. LCN lesions have to be suspected if there are typical symptoms or signs attributable to a LCN. Causes of LCN lesions can be classified as genetic, vascular, traumatic, iatrogenic, infectious, immunologic, metabolic, nutritional, degenerative, or neoplastic. Treatment of LCN lesions depends on the underlying cause. An effective treatment is available in the majority of the cases, but a prerequisite for complete recovery is the prompt and correct diagnosis. LCN lesions need to be considered in case of disturbed speech, swallowing, coughing, deglutition, sensory functions, taste, or autonomic functions, neuralgic pain, dysphagia, head, pharyngeal, or neck pain, cardiac or gastrointestinal compromise, or weakness of the trapezius, sternocleidomastoid, or the tongue muscles. To correctly assess manifestations of LCN lesions, precise knowledge of the anatomy and physiology of the area is required.

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Síndrome de Ramsay-Hunt causante de parálisis laríngea

Cited by 5 publications

References 10 publications

Ramsay Hunt syndrome and zoster laryngitis with multiple cranial nerve involvement

Ramsay Hunt syndrome and zoster laryngitis with multiple cranial nerve involvement

Síndrome de Ramsay Hunt: revisión narrativa

Disorders of the lower cranial nerves

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