Significance of elevated MB creatine kinase in patients after cardiac catheterization

Rosenblum, Andrew M.; Ludbrook, Philip A.; Jaffe, Allan S.

doi:10.1002/ccd.1810100604

Cited by 5 publications

(2 citation statements)

References 11 publications

(4 reference statements)

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Most patients survive MI without further complication, although urgent coronary artery bypass grafting may be necessary. Although total creatine kinase (CK) usually increases during cardiac catheterization as a result of soft-tissue trauma during the catheterization, the CK MB fraction should not significantly increase [44,45]. Therefore, the CK MB fraction remains a reliable test when MI is considered as a complication of coronary angiography.…”

Section: Coronary Angiography and Uentriculographymentioning

confidence: 98%

Iatrogenic Cardiovascular Disease Secondary to Diagnostic and Therapeutic Procedures

Gold

Rios²

1987

J Intensive Care Med

View full text Add to dashboard Cite

Gold RL, Rios JC. latrogenic cardiovascular disease secondary to diagnostic and therapeutic procedures. J Intensive Care Med 1987;2:49-60.The number of diagnostic and therapeutic procedures performed in cardiology continues to grow. These procedures are generally considered safe or of minimal risk to the patient. However, it is important to remember that significant complications may occur, and in each patient the risk: benefit ratio must be carefully weighed.In this review, the complications documented in the medical literature resulting from the use of cardiologic interventions and procedures are discussed. A thorough knowledge of these complications and their precipitating factors can help minimize the risk to the patient.

show abstract

Section: Coronary Angiography and Uentriculographymentioning

confidence: 98%

Iatrogenic Cardiovascular Disease Secondary to Diagnostic and Therapeutic Procedures

Gold

Rios²

1987

J Intensive Care Med

View full text Add to dashboard Cite

show abstract

“…tions [5]. MB CK elevations have been reported after PTCA in patients with overt infarction [l] (eg, totally blocked arteries, coronary dissection, severe chest pain with ECG changes), and infarction resulting from side branch occlusion [6].…”

Section: Introductionmentioning

confidence: 99%

Paucity of subtle myocardial injury after angioplasty delineated with MB CK

Spadaro

Ludbrook

Tiefenbrunn

et al. 1986

Cathet. Cardiovasc. Diagn.

Self Cite

View full text Add to dashboard Cite

To determine the extent to which PTCA elicits subtle myocardial ischemic injury, 28 patients were studied prospectively. The presence of myocardial injury was assessed by analysis of frequent (q4 hours) serial samples of MB CK, a sensitive and specific marker of myocardial injury. Immediately prior to angioplasty, baseline 12-lead ECGs and blood samples for evaluation of total CK and MB CK were obtained. Additional ECGs were obtained immediately after the procedure and on the following morning, and serial samples for total and MB CK were obtained at 4-hour intervals for 24 hours. Patients requiring cardiac surgery within 24 hours after PTCA were excluded. Samples (n = 203) were assayed by the glass bead method. Total CK rose by more than 20 IU/L in only five patients, four of whom had received intramuscular premedication. Mean MB CK was 3.4 +/- 6.1 IU/L at 12 hours and 4.1 +/- 4.7 IU/L at 24 hours compared to 3.1 +/- 1.7 IU/L prior to angioplasty. Only one patient with obvious infarction had a value for MB CK above the normal range. Thus, uncomplicated PTCA is not accompanied by objectively detectable subtle myocardial injury.

show abstract