In 1875 Richard Caton observed electrical oscillations from the brains of animals which altered during dying and ceased with death. Similar alterations may follow cerebral circulatory arrest, exsanguination or inadequate cardiac action. The EEG fails within 20 s of cessation of the circulation and can recover within 30 s of its return, but there is a longer latency to return of EEG if ischaemia is more than momentary. The decline of the EEG was considered to be characterized by a decrease in the faster waves and an increase in the slower ones leading to electrical silence, with a reversal of this sequence during recovery (fig. 1). However, with abrupt onset of ischaemia rapid transition from a continuous "activated" EEG to a burst suppression pattern and silence is more often observed (Brierley et al., 1978). The EEG changes and their time course are comparable in both localized and global ischaemia, and similar but totally reversible depression is also/ound when brain metabolism is depressed by narcdsis or profound hypothermia (Prior, 1973).