Sensory Guillain-Barré syndrome: A case report

Zhang, Jing; Liu, Na; Zhang, Zhe‐Cheng; Zheng, Ruizhi; Li, Qian

doi:10.3892/etm.2014.1995

Cited by 3 publications

(2 citation statements)

References 13 publications

(11 reference statements)

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“…The lack of formal diagnostic criteria is problematic because authors characterise the cases as sensory GBS, but the clinical presentations are heterogeneous, including descriptions of trigeminal nerve involvement,10 a case with normal reflexes11 and a number of cases with normal nerve conduction studies that were characterised as possible small fibre variants 12 13…”

Section: Discussionmentioning

confidence: 99%

Unusual sensory variant of Guillain-Barré syndrome

et al. 2017

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We describe a 52-year-old woman presenting with acute onset of severe burning paraesthesia in the hands and feet associated with allodynia and antalgic gait. At the time of admission to hospital no motor weakness was present. A diagnosis of Guillain-Barré syndrome (GBS) was considered when neurophysiological studies were completed showing convincing evidence of demyelination on motor conduction studies and sural sparing on sensory nerve studies.1 We describe this case as a sensory variant of GBS. Clinical improvement followed treatment with a single course of intravenous immunoglobulin (IVIG). The patient made a complete clinical recovery within 6 months of onset and repeat neurophysiological studies showed marked improvement. We encourage clinicians to consider an atypical variant of GBS in patients presenting with acute sensory complaints.

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Section: Discussionmentioning

confidence: 99%

Unusual sensory variant of Guillain-Barré syndrome

et al. 2017

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“…Guillain-barre syndrome (GBS) is an acute immune-mediated polyneuropathy that usually presents with progressive ascending weakness [1,2]. It has been recently understood that an antecedent infection may lead to the development of GBS.…”

Section: Introductionmentioning

confidence: 99%

The Development of Guillain-Barre Syndrome (Gbs) in Association with Confirmed Lyme Disease. A Potential Autoimmune Response in Gbs Secondary to Tick-Borne Diseases?

Awan¹

2015

Clin Microbiol

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Vector borne diseases have been associated with a number of autoimmune diseases. In this case we report a 73 year old woman initially diagnosed with lyme disease who subsequently developed guillain-barre syndrome (GBS). Her relevant clinical presentation included protean symptoms of numbness and progressive weakness in her legs for three weeks prior to consultation by the neurology service. A lyme ELISA and confirmatory Western Blot testing were unequivocally positive. A clinical diagnosis of CNS lyme disease was made. Additional clinical evaluation included: electrophysiological testing, which demonstrated significant polyneuropathy consistent with demyelinating pathology. A lumbar puncture with cerebral spinal fluid analysis revealed a non-reactive VDRL, negative lyme DNA PCR, positive lyme IgG antibody and an elevated protein-elevated albumin with normal white count. Upon further clinical deliberation this constellation of signs and symptoms was determined to be more consistent with a diagnosis of GBS rather than CNS lyme disease. Initiation of medical therapy included IVIG (intravenous immunoglobulin) and parenteral ceftriaxone. This case may report an additional vector of disease in patients presenting with clinical signs and symptoms of GBS.

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