Selective denervation of the S-A node in the treatment of progressive central vagal bradycardia

Coryllos, E; Delaney, Thomas J.; Reitman, Milton J.; Mohtashemi, M; Kenigsberg, Kenneth

doi:10.1016/s0022-3468(84)80274-2

Cited by 3 publications

(1 citation statement)

References 21 publications

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“…The medical treatment of these symptomatic bradycardia has led to numbers of studies [51][52][53] which have not always been conclusive. Therefore, some groups have tried pacemaker implantation [54][55][56][57] or even surgical cardiac denervation [58]. The efficiency of an atropine derivative has been evaluated in a multicentric French trial which started in 1988 [59] and was updated in 1995 [60] before the important changes in SIDS incidence following changing sleep position: there were 9,091 infants including 2,840 SIDS siblings.…”

Section: Treatment Justificationmentioning

confidence: 99%

Paroxysmal Vagal Overactivity, Apparent Life-Threatening Event and Sudden Infant Death

Lucet¹,

Bethmann

Denjoy³

2000

Neonatology

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The possibility of reversible cardiac asystoly due to paroxysmal vagal overactivity (VO) has been well studied, first in adults, then in children, and finally in breath-holding spells. Few studies deal with infants and the incidence of VO among preterm, apparent life-threatening event (ALTE) and sudden infant death syndrome (SIDS). In this review, we summarize data acquired during the past 20 years leading to the diagnosis of VO in infants. We describe the clinical aspects of VO in infants and young children such as Holter-ECG and oculocardiac reflex criteria. The familial aspect of VO and its possible link with SIDS (a new risk factor?) are discussed. Finally, whether or not to treat infants with symptomatic VO is discussed.

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