“…Various etiological factors for SAA have been reported, including angiodysplasia [6], segmental arterial mediolysis [18], portal hypertension [6,8], pregnant or multiparous woman [4,6], atherosclerosis [6,8], essential hypertension [8,19], diabetes mellitus [19], syphilis [6], intracranial aneurysm [19], embolic diseases [6], polyarteritis nodosa [19], α1-antitrypsin deficiency [20], and infective factors and inflammation [6,8]. Interestingly, although the SAA in this case had severe calcification, the pathology evaluation revealed no atherosclerotic changes.…”