Schistosomiasis japonica of the liver: contrast-enhanced CT findings in 113 patients.

Immunopathogenesis of human schistosomiasis

“…As well, the much higher egg production by S. japonicum (10 times that of S. mansoni ) results in increased pathology (3). The smaller size of S. japonicum eggs allows them to be swept to the small portal veins and causing fibrosis in both the peripheral and central areas of the liver (50,51), whereas S. mansoni eggs remain in the large portal veins and cause fibrosis in the central part of the organ (50,51). Calcification of S. japonicum eggs is common but occurs rarely for S. mansoni eggs (28,52).…”

Ultrasound of diffuse liver disease and portal hypertension

“…At CT, different features have been described in patients with schistosomiasis japonicum: septal calcifications; capsular calcifications; septal enhancement; and capsular enhancement [10]. Magnetic resonance imaging is not as interesting as CT because the characteristic calcifications are not seen; however, MR images show the septa as linear abnormalities that have low signal intensity on T1-and variable signal intensity on T2-weighted images [10].…”

“…On color Doppler, the left branch of the portal vein is patent, but partial obstruction due to clotting is seen laterally frequent collaterals detected are the gastroesophageal veins, the paraumbilical vein, and the splenorenal or gastrorenal veins (Figs. 8,9,10). There is no relation between the collateral pathways and the location and causes of the portal hypertension except the paraumbilical vein.…”

“…In compensated patients with small varices, endoscopy should be repeated at 1-to 2-year intervals to evaluate progression of varices 10.…”

Clinical relevance of radiographic linear branching mineral opacities in the canine liver

“…Parenchymal mineralisation may also occur following chronic conditions such as chronic hepatopathy or parasitic infection (e.g. schistosomiasis, histoplasmosis, fascioliasis, echinococcosis) (Grange et al 1974, Monzawa et al 1993, Pan et al 1999, Polat & Atamanalp 2009), bacterial infections (e.g. tuberculosis) (Maglinte et al 1988, Wong & Ng 1993, Sheen-Chen et al 2001, viral infections (Konen et al 2000, Saikia et al 2007 or in diseases causing tissue necrosis such as neoplasia (Shapiro et al 1988, Mitsudo et al 1995, Nagakura et al 1999, Murakami et al 2013, Bayraktutan et al 2014, Wang et al 2014, Inoko et al 2015, granulomas (Akimoto et al 1993), haematomas or abscesses (Reeder 1975, Paley & Ros 1998, Stoupis et al 1998.…”