Role of blood cultures during continuous renalreplacement theraphy in septic patients

Naharro, JA Monsalve; Cuesta-Montero, Pablo; Domingo-Chiva, Esther; Paniagua, S Plata; López-Pérez, Alfonso; Martínez-López, Fernando; Jimenez-Vizuete, JM

doi:10.1186/2197-425x-3-s1-a59

Cited by 1 publication

(1 citation statement)

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“…40,41,42 Pulmonary autopsy in severe and critical clinical COVID-19 shows fibrin deposition, diffuse alveolar damage, thickening of blood vessel walls, and frequent blockage of pulmonary capillaries by microthrombi and larger thrombi causing pulmonary artery thrombosis and pulmonary embolism. 43 The hypercoagulable state in COVID-19 patients causes ventilation-perfusion imbalance (V/Q mismatch) and lung tissue damage. In addition, coagulation can also activate C-reactive protein (CRP) and activate subsequent complement and fibrinogen synthesis in the liver as an acute-phase protein in COVID-19.…”

Section: Intravascular Microthrombusmentioning

confidence: 99%

Happy Hypoxemia In COVID-19

2022

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The new coronavirus disease 2019 (COVID-19) pandemic is becoming a global health crisis that challenges health systems around the world. The clinical spectrum of COVID-19 varies from mild upper respiratory tract infections to severe pneumonia and acute respiratory distress syndrome, but there are also many patients present with an SpO2 < 90% but without symptoms of shortness of breath. This phenomenon is called happy hypoxemia or silent hypoxemia. Shortness of breath or dyspnea is a subjective sensation resulting from inadequate breathing effort and is usually described as constriction in the chest, difficulty breathing air or difficulty breathing. Breathing is controlled centrally by the respiratory center in the medulla oblongata and the pons region which controls the "respiratory drive" and adapts respiration to the body's metabolic needs. The mechanisms that play a role in this phenomenon include the presence of intrapulmonary shunts, loss of pulmonary perfusion regulation, vascular microthrombus and impaired pulmonary diffusion capacity.

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