Risk of Retinal Vein Occlusion With Central Serous Chorioretinopathy

Chang, Yuh‐Shin; Chang, Chi-Lung; Weng, Shih‐Feng; Wang, Jhi-Joung; Jan, Ren‐Long

doi:10.1097/iae.0000000000000796

Cited by 11 publications

(9 citation statements)

References 31 publications

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“…29 2.30), 32 and retinal vein occlusion (AHR ¼ 3.15, 95% CI ¼ 1.91-5.21). 33 Although no adequate explanation of the relationship between CSC and these cardiovascular diseases was identified in these previous studies, the results of the current study provide a possible explanation.…”

Section: Discussionmentioning

confidence: 52%

Peripheral Vascular Endothelial Dysfunction in Central Serous Chorioretinopathy

Wang

et al. 2017

Invest. Ophthalmol. Vis. Sci.

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Section: Discussionmentioning

confidence: 52%

Peripheral Vascular Endothelial Dysfunction in Central Serous Chorioretinopathy

Wang

et al. 2017

Invest. Ophthalmol. Vis. Sci.

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“…Central Serous Chorioretinopathy (CSCR) is idiopathic and often recurs. However, the mechanism of CSCR is unclear and it's reported that CSCR patients (>40 years) increased the risk of RVO ( 125 , 126 ). Regular review of CSCR patients may be the main approach to prevent the occurrence of RVO.…”

Section: Risk Factorsmentioning

confidence: 99%

Review: The Development of Risk Factors and Cytokines in Retinal Vein Occlusion

et al. 2022

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Retinal vein occlusion (RVO) is the second most prevalent retinal disease. Despite this, the pathogenic mechanisms and risk factors are not entirely clear. In this article, we review recent publications on the classification, pathogenesis, risk factors, ischemic changes, cytokines, and vital complications of RVO. Risk factors and cytokines are important for exploring the mechanisms and new treatment targets. Furthermore, risk factors are interrelated, making RVO mechanisms more complex. Cytokines act as powerful mediators of pathological conditions, such as inflammation, neovascularization, and macular edema. This review aims to summarize the updated knowledge on risk factors, cytokines of RVO and signaling in order to provide valuable insight on managing the disease.

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“…Therefore, several cohort studies have involved investigating the association between CSC and systemic diseases related with endothelial dysfunction. [24][25][26] Among them, a population-based cohort study demonstrated that CSC increases the risk of retinal vein occlusion. 26 Moreover, those authors emphasized that the relationship between CSC and retinal vein occlusion is based on inflammation processes.…”

Section: Discussionmentioning

confidence: 99%

“…[24][25][26] Among them, a population-based cohort study demonstrated that CSC increases the risk of retinal vein occlusion. 26 Moreover, those authors emphasized that the relationship between CSC and retinal vein occlusion is based on inflammation processes. Nitric oxide production can be disrupted by inflammation processes in CSC and subsequently result in endothelial dysfunction, which indicates an association between CSC and retinal vein occlusion.…”

Section: Discussionmentioning

confidence: 99%

Evaluation of Retinal Vessel Diameters in Eyes With Active Central Serous Chorioretinopathy

Altunel

Arifoğlu

2021

Retina

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Purpose: To compare the retinal vessel diameters of healthy eyes and active central serous chorioretinopathy (CSC) eyes, and to evaluate possible effect of retinal vessel diameter alterations on the pathogenesis of CSC.Methods: This retrospective study included 39 patients with CSC and 34 healthy individuals. Spectralis optical coherence tomography + HRA with an infrared reflectance image were used to evaluate structure of retinal vessels in the circular region around the optic disc. For each eye, vertical inner and outer diameters of the four major arteries and veins were measured using infrared reflectance images, and vessel wall thicknesses were also calculated based on inner and outer diameters.Results: The 304 vessels of the 39 active CSC eyes and 266 vessels of the 34 healthy eyes were used in the analyses. The mean venous wall thickness in active CSC eyes was significantly thicker than that in healthy eyes (40.0 ± 4.9 vs. 33.5 ± 4.1 mm, P = 0.001), whereas the mean venous inner diameter in active CSC eyes was significantly narrower (52.5 ± 9.7 vs. 61.3 ± 8.1 mm, P = 0.001). Also, the mean venous outer diameter was wider in CSC eyes, albeit not significantly (131.1 ± 7.0 vs. 128.5 ± 8.4 mm, P = 0.074).Conclusion: Our results suggest that the alterations of retinal venous diameters may play a potential role in the pathogenesis of CSC in addition to alterations in choroidal thickness.

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Risk of Retinal Vein Occlusion With Central Serous Chorioretinopathy

Abstract: Central serous chorioretinopathy increases the risk of RVO. For CSCR patients, the authors recommend thorough retinal vessel evaluation, regular follow-up, and education regarding RVO for patients with CSCR.

Cited by 11 publications

References 31 publications

Peripheral Vascular Endothelial Dysfunction in Central Serous Chorioretinopathy

Peripheral Vascular Endothelial Dysfunction in Central Serous Chorioretinopathy

Review: The Development of Risk Factors and Cytokines in Retinal Vein Occlusion

Evaluation of Retinal Vessel Diameters in Eyes With Active Central Serous Chorioretinopathy

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