2014
DOI: 10.1177/1470320314563424
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RETRACTED: Role of renin-angiotensin-aldosterone system inhibitors in radiation nephropathy

Abstract: The following article has been included in a multiple retraction: Tian-Biao Zhou, Hong-Yan Li, Zong-Pei Jiang, Jia-Fan Zhou, Miao-Fang Huang and Zhi-Yang Zhou Role of renin-angiotensin-aldosterone system inhibitors in radiation nephropathy Journal of Renin-Angiotensin-Aldosterone System (JRAAS) 1470320314563424, first published 18 December 2014. DOI: 10.1177/1470320314563424. This article has been retracted at the request of the Editors and the Publisher. After conducting a thorough investigation, SAGE found t… Show more

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Cited by 2 publications
(2 citation statements)
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“…Kidneys are key players in regulating systemic blood pressure through the renin-angiotensin-aldosterone system (RAAS). It is well established that RAAS-inhibition via the angiotensin-converting enzyme (ACE) and angiotensinogen I (ATI) inhibitors alleviates the progression of many kidney diseases including radiation nephropathy (37). Elevated diastolic and systolic blood pressures were noted after 100 days of kidney irradiation in mice, which were further increased upon bilateral renal radiation, pointing out that radiation nephropathy has long-lasting functional and metabolic consequences (38).…”
Section: Discussionmentioning
confidence: 99%
“…Kidneys are key players in regulating systemic blood pressure through the renin-angiotensin-aldosterone system (RAAS). It is well established that RAAS-inhibition via the angiotensin-converting enzyme (ACE) and angiotensinogen I (ATI) inhibitors alleviates the progression of many kidney diseases including radiation nephropathy (37). Elevated diastolic and systolic blood pressures were noted after 100 days of kidney irradiation in mice, which were further increased upon bilateral renal radiation, pointing out that radiation nephropathy has long-lasting functional and metabolic consequences (38).…”
Section: Discussionmentioning
confidence: 99%
“…Previous studies demonstrated that storax could attenuate the brain damage and regulate the coagulation function in acute focal cerebral ischemia in rats. Storax significantly attenuated the infarct volumes, hemispheric swelling rates, and neurological deficits; decreased the fibrinogen content; prolonged the prothrombin time; and activated the partial thromboplastin time and the content of nitric oxide in serum in focal ischemic stroke rats (Ni et al., 2011a; Zhou et al., 2015). Storax exerts protective effects on astrocytes and the blood brain barrier in ischemia-reperfusion injury after stroke (Ni et al., 2011b).…”
Section: Introductionmentioning
confidence: 99%