Results in 208 Iris-Clip Pseudophakos Implantations

Binkhorst, C.D.; Leonard, Paul

doi:10.1016/0002-9394(67)92241-6

Cited by 34 publications

(7 citation statements)

References 8 publications

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“…The incidence of corneal oedema reported in these studies ranges from a few per cent to 13%. Other investigators have failed to demonstrate this higher incidence (Binkhorst & Leonard 1967).…”

mentioning

confidence: 83%

Corneal Thickness and Endothelial Damage After Intraocular Lens Implantation

Olsen

Eriksen

1980

Acta Ophthalmologica

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The corneal thickness and the specular appearance of the corneal endothelium are reported in 100 patients with unilateral intraocular lens implantation. Post-operative time ranged from one to 42 months. An average central endothelial cell loss of uncomplicated cases of 46%, range 1 to 83%, with no correlation with time after the operation was found. A significantly higher cell loss was found in cases with technical complications, shallow anterior chamber or increased intraocular pressure post-operatively. No correlation was found between the corneal thickness and the endothelial cell loss. In two patients, however, with a cell density below 500 cells/mm2, a slight increase in corneal thickness was noted. Thirty patients presented a guttate endothelium. Irrespective of the occurrence of surgical complications the presence of a guttate endothelium was found to be a major determinant of the corneal thickness increase and could be ascribed as a cause of persistent corneal swelling in six of twelve patients with elevated corneal thickness. The progression of guttate changes occurred independently of the cell loss.

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confidence: 83%

Corneal Thickness and Endothelial Damage After Intraocular Lens Implantation

Olsen

Eriksen

1980

Acta Ophthalmologica

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“…The incidence of cystoid oedema of the macula in pseudophakia has been variously stated to be 5% (Pearce, 1972), 12% (Binkhorst andLeonard, 1967), 19% (Jardine andSandford-Smith, 1974), and 7% (Dallas, 1976), which are all greater than the 2% incidence following plain intracapsular cataract extraction in a series of 200 patients. Since the frequency and severity of cystoid macular oedema are known to be greater in patients with implants than in aphakic patients without implants, the question arises whether the implant is responsible.…”

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confidence: 97%

Aphakic macular oedema following prosthetic lens implantation.

Easty¹,

Dallas²,

O’Malley³

1977

British Journal of Ophthalmology

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SUMMARY Fluorescein angiography of the iris was performed on patients with plastic lens implants with cystoid oedema of the macula, and the nature of the vascular changes was compared with controls provided by patients who did not have macular disease. Densitometry was used to quantitate leakage of fluorescein dye into the anterior chamber in residual angiograms 5 minutes after injection. Cystoid oedema was associated with marked increase in vascular permeability of the iris in the patients with implants, and in aphakics without implants, but to a lesser degree. Although it cannot be concluded that implants themselves cause either the increase in permeability or the onset of macular oedema, the presence of such implants must be considered an additional hazard in an eye which is already at risk. Iris angiography can be used as a diagnostic aid in patients in whom macular oedema is suspected. A persistent increase in permeability associated with a reduction in visual acuity should act as a warning of further visual loss and of eventual cystoid macular degeneration.With the increasing use of prosthetic lens implantation following cataract extraction, the nature and incidence of operative and postoperative complications are being investigated in more depth. Postoperative anterior uveitis, retinal detachment, acrylic lens dislocation, glaucoma, bullous keratopathy, and cystoid macular oedema have all been reported, but it is the macular disease which is particularly a cause for disappointment to the patient and concern for the ophthalmic surgeon.Macular oedema and visual loss following cataract extraction were originally described by Irvine (1953) as being associated with irritability of the eye and also rupture of the anterior hyaloid membrane leading to vitreous strands adhering to the section. Gass and Norton (1969) stated that adherence and traction were not a necessity for the development of macular oedema but when present tended to worsen the condition. In a study of macular disturbance 6 weeks after cataract extraction (Hitchings et al., 1975) transient oedema occurred in 50% of patients examined. No such changes developed in a group of controls who had glaucoma surgery. The authors

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“…This is also a complication which may be related to the trauma of additional steps entailed in implant surgery. Binkhorst and Leonard (1967) reported initial results in 124 eyes, treated over a period of 7 years and did not observe a single case of intractable corneal oedema. Jardine and Sandforth-Smith (1974) had reported an incidence of 10 % in 70 cases.…”

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confidence: 99%

Endothelial cell loss and corneal thickness after intracapsular extraction and iris clip lens implantation: a randomised controlled trial (interim report).

Cheng¹,

Sturrock²,

Rubinstein³

et al. 1977

British Journal of Ophthalmology

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SUMMARY Patients in a randomised controlled trial were chosen either to have iris clip lens implantation after intracapsular cataract extraction or intracapsular extraction only. They were assessed in terms of corneal thickness, postoperative epithelial oedema, and endothelial cell counts. All patients had 1 eye submitted to operation, which was carried out by the same surgeon. There was significantly greater increase in corneal thickness (P <0-05) on the 5th postoperative day in eyes which had lens implants (23 patients with intracapsular extraction and 19 with implant), but the difference between the 2 groups became insignificant at 1 month (17 patients in each group).Daily corneal thickness measurements and observations of epithelial oedema in a subgroup (20 patients divided equally into 2 groups) showed that postoperatively there was greater and more widespread corneal oedema after implant surgery. When the operated eye was compared with the unoperated eye, endothelial cell loss was significantly greater in those with implants (P<0-01) than in those with simple intracapsular extraction.Intraocular lens implantation after cataract exti-action has regained general acceptance and is now widely practised. The reported incidence of various complications differs considerably, but the incidence of intractable corneal oedema would seem to be greater than expected from simple cataract extraction. This is also a complication which may be related to the trauma of additional steps entailed in implant surgery. Binkhorst and Leonard (1967) reported initial results in 124 eyes, treated over a period of 7 years and did not observe a single case of intractable corneal oedema. Jardine and Sandforth-Smith (1974) had reported an incidence of 10 % in 70 cases. Pearce (1972, 1975), Jaffe and Duffner (1976), and Duffner et al. (1976) have reported an incidence of between 1P5 and 36y% of serious corneal oedema. Pearce (1975) also reported an incidence of 13 2y% of minor oedema which had not interfered with vision in the eyes studied.

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Results in 208 Iris-Clip Pseudophakos Implantations

Cited by 34 publications

References 8 publications

Corneal Thickness and Endothelial Damage After Intraocular Lens Implantation

Corneal Thickness and Endothelial Damage After Intraocular Lens Implantation

Aphakic macular oedema following prosthetic lens implantation.

Endothelial cell loss and corneal thickness after intracapsular extraction and iris clip lens implantation: a randomised controlled trial (interim report).

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