Respiratory Function in Infants With Arnold???chiari Malformation

Krieger, Abbott J.; Detwiler, John S.; Trooskin, Z B S Stanley

doi:10.1288/00005537-197605000-00013

Cited by 20 publications

(3 citation statements)

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“…Laryngeal stridor due to vocal fold paralysis associated with Chiari II malformations (also known as Arnold-Chiari malformations) has been commonly reported. [2][3][4][5][6][7][8][9] There have been few reported cases of laryngomalacia associated with Chiari malformations, and no reported cases of laryngomalacia caused by C1 posterior arch compression of the cervicomedullary junction. Portier et al 6 described a case of an infant with a Chiari I malformation (elongation of cerebellar tonsils through the foramen magnum) and laryngomalacia causing respiratory obstruction that resolved 10 days after posterior fossa decompression.…”

Section: Discussionmentioning

confidence: 99%

“…1 Laryngeal stridor has been reported in association with Chiari II malformations (caudal displacement of the brain stem into the cervical spinal canal associated with myelomeningocele), with the stridor most often caused by vocal fold paralysis. [2][3][4][5][6][7][8][9] In some of these cases, the actual laryngeal abnormality was not discovered. 3,4 We present 2 cases of infants with neurologic variant laryngomalacia.…”

Section: Introductionmentioning

confidence: 99%

“…[2][3][4][5][6][7][8][9] In some of these cases, the actual laryngeal abnormality was not discovered. 3,4 We present 2 cases of infants with neurologic variant laryngomalacia. One infant was found to have an underlying Chiari I malformation, and the other was found to have cervicomedullary compression by a C1 posterior arch.…”

Section: Introductionmentioning

confidence: 99%

See 2 more Smart Citations

Neurologic Variant Laryngomalacia Associated with Chiari Malformation and Cervicomedullary Compression: Case Reports

Petersson

Wetjen

Thompson

2011

Ann Otol Rhinol Laryngol

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Two infants presented with intermittent stridor and evidence of laryngomalacia on flexible laryngoscopy. The first was a 10-month-old girl who had undergone 3 supraglottoplasty surgeries at an outside institution, without long-term resolution of symptoms. She was found during our evaluation to have a Chiari malformation. Laryngomalacia symptoms resolved after suboccipital decompression and C1 laminectomy, and the patient remained symptom-free at 6-month follow-up. The second infant was a 24-day-old boy with velocardiofacial syndrome who was found to have posterior cervicomedullary junction compression at the level of C1. He underwent C1 laminectomy for decompression of the brain stem, which resulted in immediate resolution of symptoms, and he remained symptom-free at 12-month follow-up. Neurologic abnormalities have been reported in up to 50% of infants with laryngomalacia. As such, brain stem dysfunction should be considered among the causes of laryngomalacia during evaluation, especially in patients with failure of supraglottoplasty. Both of these infants had resolution of symptoms after their neurosurgical procedures.

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