2005
DOI: 10.1055/s-2005-915597
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Resolution of Trigeminal Neuralgia Following Third Ventriculostomy for Hydrocephalus Associated with Chiari I Malformation: Case Report

Abstract: This is the first case in which trigeminal neuralgia was treated with a third ventriculostomy and one of only four cases of isolated trigeminal neuralgia associated with a Chiari malformation. Acquired aqueductal stenosis may have caused the hydrocephalus which, in turn, caused the Chiari malformation configuration that caused the trigeminal neuralgia. The rationale for the treatment modality and possible causes of Chiari I-induced trigeminal neuralgia are discussed.

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Cited by 21 publications
(16 citation statements)
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“…1,3,11,[13][14][15][16][17] It has been more commonly linked to syringobulbia associated with a variety of hindbrain malformations. 8 As demonstrated in the imaging in this particular patient, there was no evidence of syringobulbia or hydrocephalus to explain the symptoms of facial pain.…”
Section: Discussionmentioning
confidence: 99%
“…1,3,11,[13][14][15][16][17] It has been more commonly linked to syringobulbia associated with a variety of hindbrain malformations. 8 As demonstrated in the imaging in this particular patient, there was no evidence of syringobulbia or hydrocephalus to explain the symptoms of facial pain.…”
Section: Discussionmentioning
confidence: 99%
“…9 Another theory is that a cervical syrinx may cause indirect vascular compression or stretching or the trigeminal nerve at the nerve root entry zone. 12 Finally, the Chiari malformation may cause micro-ischemic changes, which have been shown to cause trigeminal neuralgia in association other lesions. 8 Approximately two-thirds of the patients described in the literature had sustained pain relief following Chiari decompression.…”
Section: 15mentioning
confidence: 99%
“…К та-ким заболеваниям относятся: опухоли мосто-мозжескового угла [67,145,181,211,244,248,287,319,347,348], арахноидиты задней че-репной ямки [142,323], аневризмы и артерио-венозные мальфор-мации вертебробазилярного бассейна [150,240,254,265,309,361,352,425], аномалии Арнольда-Киари [218,405], платибазии [264,357]. Клинически этот вид НТН отличается от классической нарастающим сенсорным дефицитом, не характерным для клас-сической НТН.…”
Section: от истоков к современному взгляду на патогенезunclassified