Resistência à proteína C ativada e doença arterial isquêmica em jovem

Menezes, Igor Alexandre Côrtes de; Romani, Rafael Fernandes; Aoki, Yuki; Abdo, João Caetano; Carvalho, Mauricio

doi:10.1590/s0066-782x2010000300025

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2013

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“…6,7 Inherited or acquired defects of coagulation system that cause hypercoagulability defined as thrombophilia including the anticardiolipin antibody, lupus anticoagulant, protein C and S deficiencies, factor V Leiden (FVL) and the G20210A mutation of the prothrombin gene. 9 The FVL is reported as the most common thrombophilia with a prevalence of 5% among the Caucasian population. The activated factor V acts as a cofactor to activated factor X in the conversion of prothrombin to thrombin and its function is down-regulated by the activated protein C (APC) for haemostatic maintenance.…”

Section: Casementioning

confidence: 99%

Keeping an Open Mind about Acute Flank Pain in Emergency Department: Acute Kidney Infarction

Bacaksız¹,

Erdoğan²,

Tasal³

et al. 2013

Hong Kong j. emerg. med.

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Differential diagnosis of the acute flank pain in a patient without nephrolithiasis is challenging in emergency department. Renal infarction should be suspected if accompanying cardiac arrhythmia, heart failure or valvular disease is present. Activated protein C resistance is the most common thrombophilia that triggers venous-obstructive conditions and rarely arterial thromboembolism. We described a young male with a history of non-ischemic dilated cardiomyopathy and coexisting activated protein C resistance presented with new onset acute flank pain due to acute renal infarction.

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Section: Casementioning

confidence: 99%