“…Offspring quality and quantity will depend on fertility, fecundity, and development (viability) of those offspring, each of which are influenced by diet. Equivalent changes in these traits can be elicited in distinct taxa by equivalent dietary changes: for example, in numerous studies of insects and mammals, amino acid nutrition has proven consistently important for fertility, fecundity and offspring development (Bong et al, 2014; Crean and Senior, 2019; Dong et al, 2016; Fanson et al, 2012; Grandison et al, 2009; Jensen et al, 2015; Lee et al, 2008; Leitão-Gonçalves et al, 2017; Liang and Zhang, 2006; Ma et al, 2020; Maklakov et al, 2008; McCracken et al, 2020; Mirth et al, 2018; Skorupa et al, 2008; Solon-Biet et al, 2015; Svajgr et al, 1972; Tufarelli et al, 2015; Winship et al, 2018; Wong et al, 2014a; Zanco et al, 2020); whereas high-lipid or high-sugar diets are obesogenic (Dobson et al, 2019; Hariri and Thibault, 2010; Jang et al, 2018; Rivera et al, 2015; Saud et al, 2015; Wong et al, 2014b; Woodcock et al, 2015; Zhao et al, 2020). However, importantly, reproduction is not only regulated by the individual’s current diet: large nutritional fluctuations can induce enduring physiological and molecular changes, which can transmit across generations, influencing quality of offspring and even grand-offspring (Barker and Osmond, 1986; Barker and Thornburg, 2013; Deas et al, 2019; Duque-Guimarães and Ozanne, 2017; Holland et al, 2016; Li et al, 2018; Liang and Zhang, 2006; Öst et al, 2014; Rivera et al, 2015; Stefana et al, 2017; Wei et al, 2014; Winship et al, 2018).…”