Reply to Winter et al.

Ostrovsky, Olga; Korostishevsky, Michael; Nagler, Arnon

doi:10.1038/leu.2008.39

Leukemia

2008

DOI: 10.1038/leu.2008.39

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Reply to Winter et al.

Olga Ostrovsky

Michael Korostishevsky

Arnon Nagler

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2009

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“…21 Platelet-activating factor (PAF) translocated to the surface of collagen-adherent platelets mediates neutrophil activation and firm adhesion to platelet monolayers. 9 Additional adhesive and signaling mechanisms are also involved. 2,7 In addition to P-selectin/PSGL-1, other molecular interactions mediate intercellular interactions between activated platelets and myeloid leukocytes (see Figure 1).…”

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confidence: 99%

Platelet–Leukocyte Interactions in Inflammation and Thrombosis

Michetti¹,

Weyrich²,

Zimmerman³

2009

Oncology &Amp; Hematology Review (US)

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Interactions between human platelets and leukocytes occur in physiological and pathological inflammation, hemostasis, and thrombosis. There is evidence that platelet–leukocyte interactions may be central mechanisms in atherosclerosis and its complications, and also in a variety of other acute and chronic diseases. In addition, models based on interactions of platelets and leukocytes are informative discovery tools that have yielded new insights relevant to human cell functions and cell–cell signaling and information transfer. This short article focuses on interactions between platelets and myeloid leukocytes and profiles some of the key pathways, mechanisms, and outcomes in these cellular dialogues.

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mentioning

confidence: 99%

Platelet–Leukocyte Interactions in Inflammation and Thrombosis

Michetti¹,

Weyrich²,

Zimmerman³

2009

Oncology &Amp; Hematology Review (US)

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show abstract

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Reply to Winter et al.

Cited by 1 publication

References 4 publications

Platelet–Leukocyte Interactions in Inflammation and Thrombosis

Platelet–Leukocyte Interactions in Inflammation and Thrombosis

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