“…In wounds infected with P. aeruginosa , the rate of repair is diminished in multiple animal models (e.g., rabbit, murine, pig in vivo models) [ 104 ]. Specifically, in a murine wound model (in which a muscle was injured in the right rectus abdominus to which P. aeruginosa was applied), transcriptional profiling identified 7 out of 136 differentially expressed genes that were involved in pyochelin biosynthesis, including the pyochelin receptor fptA, pchH, pchG, pchE, pchD, pchB, and pchC (the biosynthesis of Pch requires the iron-regulated pchDCBA operon) [ 106 ].…”